Neutrophils in antibody-based immunotherapy of cancer
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Summary
The scientific data that supports the possibility of exploiting neutrophils for mAb-based immunotherapy of cancer is reviewed, and an increasing awareness and understanding of this topic may allow for future development of new anticancer therapies.
- Type
- article
- Published
- 2008-01-01
- Cited by
- 15
- References
- 98
- OpenAlex
- https://openalex.org/W18081538
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:43029818
Keywords
Computer science
References
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Cited by
- CD11c+ Dendritic Cells and B Cells Contribute to the Tumoricidal Activity of Anti-DR5 Antibody Therapy in Established Tumors
- Enhancement of antibody-dependent cell-mediated cytotoxicity by endowing IgG with FcαRI (CD89) binding
- Human IgG2 Antibodies against Epidermal Growth Factor Receptor Effectively Trigger Antibody-Dependent Cellular Cytotoxicity but, in Contrast to IgG1, Only by Cells of Myeloid Lineage
- Novel Antibody-Based Proteins for Cancer Immunotherapy
- Protumor and antitumor functions of neutrophil granulocytes
- Myeloid cells as effector cells for monoclonal antibody therapy of cancer.
- Targeting FcαRI on Polymorphonuclear Cells Induces Tumor Cell Killing through Autophagy
- Polymorphonuclear neutrophils and cancer: Intense and sustained neutrophilia as a treatment against solid tumors
- Diagnostic role of neutrophil-lymphocyte ratio in oral cavity cancers
- Neutrophil mediated drug delivery for targeted glioblastoma therapy: A comprehensive review.
- N1 versus N2 and PMN‐MDSC: A critical appraisal of current concepts on tumor‐associated neutrophils and new directions for human oncology
- Neutrophils in glioma microenvironment: from immune function to immunotherapy
- Behaviorome profiling of anti-tumor and pro-tumor human neutrophil subtypes in a microphysiological system.
- Antibody-Dependent Cellular Cytotoxicity Growth Factor Receptor Effectively Trigger Human IgG2 Antibodies against Epidermal
- TumorsAnti-DR5 Antibody Therapy in Established Contribute to the Tumoricidal Activity of Dendritic Cells and B Cells + CD11c
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