Effects of an unusual poison identify a lifespan role for Topoisomerase 2 in Saccharomyces cerevisiae

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Summary

Evidence is presented supporting the hypothesis that an age-associated decay in genome maintenance promotes aging in Saccharomyces cerevisiae due to an inability to sense or repair DNA damage by topoisomerase 2 (yTop2), and intrinsic yTop2-mediated DNA damage is identified as a potentially manageable cause of aging.

Type
article
Published
2017-01-01
Cited by
12
References
101
Access
Open access

Keywords

Saccharomyces cerevisiae, White (mutation), Gerontology, Art, Philosophy

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