Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network.

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Summary

A model is proposed in which the context-dependent cytostatic and promitogenic functions of specific interleukins contribute to connect senescence with an inflammatory phenotype and cancer.

Type
article
Published
2008-06-13
Cited by
2,016
References
56
Access
Open access

Keywords

Biology, Senescence, Oncogene, Interleukin, Cancer research

References

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