Role of Ca2+ signaling in skeletal muscle hypertrophy and atrophy
Explore this paper's citation graph
- Type
- article
- Published
- 2015-05-25
- Cited by
- 4
- References
- 81
- Access
- Open access
- OpenAlex
- https://openalex.org/W1821292396
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:70602218
Keywords
Muscle hypertrophy, Intracellular, Cell biology, Regulator, Skeletal muscle
References
- The E3 Ligase MuRF1 degrades myosin heavy chain protein in dexamethasone-treated skeletal muscle.
- Proteasome-dependent Activation of Mammalian Target of Rapamycin Complex 1 (mTORC1) Is Essential for Autophagy Suppression and Muscle Remodeling Following Denervation*
- Nifedipine Treatment Reduces Resting Calcium Concentration, Oxidative and Apoptotic Gene Expression, and Improves Muscle Function in Dystrophic mdx Mice
- Orexin/Hypocretin Activates mTOR Complex 1 (mTORC1) via an Erk/Akt-independent and Calcium-stimulated Lysosome v-ATPase Pathway*
- Calcium signalling: dynamics, homeostasis and remodelling
- Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways
- Localized Igf-1 transgene expression sustains hypertrophy and regeneration in senescent skeletal muscle
- Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo
- Skeletal muscle hypertrophy is mediated by a Ca2+-dependent calcineurin signalling pathway
- IGF-1 induces skeletal myocyte hypertrophy through calcineurin in association with GATA-2 and NF-ATc1
- Increase in levels of polyubiquitin and proteasome mRNA in skeletal muscle during starvation and denervation atrophy.
- The IGF-1/PI3K/Akt pathway prevents expression of muscle atrophy-induced ubiquitin ligases by inhibiting FOXO transcription factors.
- Mechanical stimuli regulate rapamycin-sensitive signalling by a phosphoinositide 3-kinase-, protein kinase B- and growth factor-independent mechanism.
- Inhibition of Atrogin-1/MAFbx Mediated MyoD Proteolysis Prevents Skeletal Muscle Atrophy In Vivo
- Vanilloid receptor expressed in the sarcoplasmic reticulum of rat skeletal muscle.
- Balancing muscle hypertrophy and atrophy
- Matching of Calcineurin Activity to Upstream Effectors Is Critical for Skeletal Muscle Fiber Growth
- Exercise metabolism and the molecular regulation of skeletal muscle adaptation.
- Amino acids activate mTOR complex 1 via Ca2+/CaM signaling to hVps34.
- BMP signaling controls muscle mass
Cited by
- Amino Acid-Mediated Intracellular Ca2+ Rise Modulates mTORC1 by Regulating the TSC2-Rheb Axis through Ca2+/Calmodulin
- Adeno-associated virus-mediated expression of an inactive CaMKIIβ mutant enhances muscle mass and strength in mice.
- Molecular Mechanisms Through Which Cannabidiol May Affect Skeletal Muscle Metabolism, Inflammation, Tissue Regeneration, and Anabolism: A Narrative Review
- The Effects of Antioxidants and Pulsed Magnetic Fields on Slow and Fast Skeletal Muscle Atrophy Induced by Streptozotocin: A Preclinical Study
Related papers
- Testosterone reduced methylprednisolone-induced muscle atrophy in spinal cord-injured rats
- Muscle Atrophy Classification: The Need for a Pathway-Driven Approach.
- Molecular Nutritional Study on Prevention of Muscle Atrophy
- Cold-induced muscle atrophy in zebrafish: Insights from swimming activity and gene expression analysis
- Exercise and Muscle Atrophy.
- A review of muscle atrophy.
- PRECLINICAL MODELS OF INACTIVITY AND BEDREST TO INVESTIGATE MUSCLE ATROPHY
- 스테로이드 유발성 근위축
- Muscle atrophy in chronic inflammatory demyelinating polyneuropathy: a computed tomography assessment
- Atrophy and hypertrophy of skeletal muscles: structural and functional aspects