Melittin-induced long non-coding RNA NONHSAT105177 inhibits proliferation and migration of pancreatic ductal adenocarcinoma
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Summary
The combined findings of the current and previous studies suggest that NONHSAT105177 mediated the melittin-induced inhibition of PDAC cell growth and metastasis, which indicated a potential target for developing new strategies.
- Type
- article
- Published
- 2018-09-20
- Cited by
- 61
- References
- 28
- Access
- Open access
- OpenAlex
- https://openalex.org/W30237397
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:52306243
Keywords
Humanities, Political science, Philosophy
References
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Cited by
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- LncRNA MIAT facilitates osteosarcoma progression by regulating mir‐128‐3p/VEGFC axis
- The functional roles of exosomal long non-coding RNAs in cancer
- Novel Molecular Targets and Mechanisms Involved in the Invasion and Metastasis of Pancreatic Cancer
- Tumor-Derived Exosomes Mediate the Instability of Cadherins and Promote Tumor Progression
- Update on current pancreatic treatments: from molecular pathways to treatment
- Insights into Biological Role of LncRNAs in Epithelial-Mesenchymal Transition
- Analysis of lncRNA–mRNA networks after MEK1/2 inhibition based on WGCNA in pancreatic ductal adenocarcinoma
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- RETRACTED: Long noncoding RNA ITGB2‐AS1 promotes growth and metastasis through miR‐4319/RAF1 axis in pancreatic ductal adenocarcinoma
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- A clinical literature review and research-trends analysis of bee venom pharmacopuncture for cancer patients
- Exosomal long non-coding RNAs: Emerging players in cancer metastasis and potential diagnostic biomarkers for personalized oncology
- Invited mini review article for the special Issue:lncRNA_precision medicine exosomal long non-coding RNAs in the diagnosis and oncogenesis of pancreatic cancer.
- KLHDC7B-DT aggravates pancreatic ductal adenocarcinoma development via inducing the crosstalk between cancer cells and macrophages.
- Extracellular vesicles as drug vectors for precise cancer treatment.
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