KLHDC7B-DT aggravates pancreatic ductal adenocarcinoma development via inducing the crosstalk between cancer cells and macrophages.

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Summary

The crosstalk between PDAC cells and macrophages induced by KLHDC7B-DT represents potential therapeutic target for PDAC.

Type
article
Published
2021-02-01
Cited by
19
References
66
Access
Open access

Keywords

Autocrine signalling, Paracrine signalling, STAT3, Cancer research, Pancreatic cancer

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