Arid5a regulates naive CD4+ T cell fate through selective stabilization of Stat3 mRNA
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Summary
It is shown that Arid5a regulates the fate of naive CD4+ T cells to pro- or antiinflammatory T cells through selective stabilization of Stat3 mRNA under Th17-polarizing conditions.
- Type
- article
- Published
- 2016-04-04
- Cited by
- 83
- References
- 53
- Access
- Open access
- OpenAlex
- https://openalex.org/W27022145
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:31184333
Keywords
Geography
References
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Cited by
- CD5: A New Partner for IL-6.
- Post-Transcriptional Gene Regulation by MicroRNA and RNA-Binding Protein
- Arid5a exacerbates IFN-γ–mediated septic shock by stabilizing T-bet mRNA
- Genome-wide map of RNA degradation kinetics patterns in dendritic cells after LPS stimulation facilitates identification of primary sequence and secondary structure motifs in mRNAs
- TLR4-induced NF-κB and MAPK signaling regulate the IL-6 mRNA stabilizing protein Arid5a
- Arid5a-deficient mice are highly resistant to bleomycin-induced lung injury
- The role and therapeutic targeting of IL-6 in rheumatoid arthritis
- Implications of IL-6 Targeting Therapy for Sepsis
- Proteolytic control of Interleukin-11 and Interleukin-6 biology.
- Interleukin (IL-6) Immunotherapy.
- Arid5a stabilizes OX40 mRNA in murine CD4+ T cells by recognizing a stem‐loop structure in its 3′UTR
- ARID5B as a critical downstream target of the TAL1 complex that activates the oncogenic transcriptional program and promotes T-cell leukemogenesis
- Regulation of inflammatory responses by dynamic subcellular localization of RNA-binding protein Arid5a
- Cytokines 2017 in Kanazawa: Looking beyond the horizon of integrated cytokine research from the sea of Japan.
- In silico analysis of expression data during the early priming stage of liver regeneration after partial hepatectomy in rat
- Accurate differential analysis of transcription factor activity from gene expression
- Posttranscriptional regulation of T helper cell fate decisions
- Post-transcriptional regulation of immune responses by RNA binding proteins
- Substrate specificity of human MCPIP1 endoribonuclease
- Chimeric Antigen Receptor-T Cells with 4-1BB Co-Stimulatory Domain Present a Superior Treatment Outcome than Those with CD28 Domain Based on Bioinformatics
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