Essential Role of Mitochondrial Antiviral Signaling, IFN Regulatory Factor (IRF)3, and IRF7 in Chlamydophila pneumoniae-Mediated IFN-β Response and Control of Bacterial Replication in Human Endothelial Cells

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Summary

It is shown that infection of human endothelial cells with C. pneumoniae-induced production of IFN-β, a cytokine that so far has been mainly associated with antiviral immunity, activates an MAVS-, IRF3-, and IRF7-dependent signaling, which controls bacterial growth and might modulate development of vascular lesions.

Type
article
Published
2010-03-15
Cited by
54
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79

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Geology

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