The role of innate immune signals in immunity to Brucella abortus
Explore this paper's citation graph
Summary
Recent advances in NOD1, NOD2, and type I IFN receptors in innate immune pathways during B. abortus infection are described, which are claimed to be the most important single TLR during Brucella infection.
- Type
- article
- Published
- 2012-10-25
- Cited by
- 56
- References
- 131
- Access
- Open access
- OpenAlex
- https://openalex.org/W23112959
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:15217222
Keywords
Pedagogy, Architectural engineering, Sociology, Engineering ethics, Computer science
References
- Essential Role of Mitochondrial Antiviral Signaling, IFN Regulatory Factor (IRF)3, and IRF7 in Chlamydophila pneumoniae-Mediated IFN-β Response and Control of Bacterial Replication in Human Endothelial Cells
- The cytosolic nucleic acid sensor LRRFIP1 mediates the production of type I interferon via a β-catenin-dependent pathway
- Role of Type I IFNs in Pulmonary Complications of Pneumocystis murina Infection 1
- Induction of Type I Interferons by Bacteria
- Type I IFNs Enhance Susceptibility to Chlamydia muridarum Lung Infection by Enhancing Apoptosis of Local Macrophages1
- Rna Polymerase III
- Central Role of MyD88-Dependent Dendritic Cell Maturation and Proinflammatory Cytokine Production to Control Brucella abortus Infection1
- Caspase-1 induced pyroptotic cell death
- Stimulator of IFN Gene Is Critical for Induction of IFN-β during Chlamydia muridarum Infection
- Interferon-induced inhibition of Chlamydia trachomatis: dissociation from antiviral and antiproliferative effects
- Bacterial RNA mediates activation of caspase-1 and IL-1β release independently of TLR3, -7, -9 and Trif but dependent on Unc93B
- The pattern recognition receptors Nod1 and Nod2 account for neutrophil recruitment to the lungs of mice infected with Legionella pneumophila.
- Sensing and Reacting to Microbes via the Inflammasomes
- Murine splenocytes produce inflammatory cytokines in a MyD88‐dependent response to Bacillus anthracis spores
- Caspase-2 Mediated Apoptotic and Necrotic Murine Macrophage Cell Death Induced by Rough Brucella abortus
- The Toll gene of Drosophila, required for dorsal-ventral embryonic polarity, appears to encode a transmembrane protein.
- Interferon: friend or foe?
- Function of Nod-like Receptors in Microbial Recognition and Host Defense
- Ubiquitylation in innate and adaptive immunity
- Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf
Cited by
- Beyond mice and men: Environmental change, immunity and infections in wild ungulates
- Toll-Like Receptor 6 Plays an Important Role in Host Innate Resistance to Brucella abortus Infection in Mice
- MyD88 knockout mice develop initial enlarged periapical lesions with increased numbers of neutrophils.
- 5-Lipoxygenase Negatively Regulates Th1 Response during Brucella abortus Infection in Mice
- TLR2 and TLR4 signaling pathways are required for recombinant Brucella abortus BCSP31-induced cytokine production, functional upregulation of mouse macrophages, and the Th1 immune response in vivo and in vitro
- Lack of Endogenous IL-10 Enhances Production of Proinflammatory Cytokines and Leads to Brucella abortus Clearance in Mice
- TLRs and tryptophan metabolism at the crossroad of immunoregulatory pathways
- Immune Responses of Bison and Efficacy after Booster Vaccination with Brucella abortus Strain RB51
- Analyzing the molecular mechanism of lipoprotein localization in Brucella
- TLR9 is required for MAPK/NF‐κB activation but does not cooperate with TLR2 or TLR6 to induce host resistance to Brucella abortus
- Structural, Functional, and Immunogenic Insights on Cu,Zn Superoxide Dismutase Pathogenic Virulence Factors from Neisseria meningitidis and Brucella abortus
- Inflammatory response of TLR4 deficient spleen macrophages (CRL 2471) to Brucella abortus S19 and an isogenic ΔmglA deletion mutant.
- NLRP12 negatively regulates proinflammatory cytokine production and host defense against Brucella abortus
- Repetitive extragenic palindromic DNA sequences from Brucella melitensis stimulate Toll-like receptor 9 signaling in macrophages.
- Clinical Use of the Interferon Inducer IIBI in Patients with Refractory Hodgkin's Lymphoma
- Getting “Inside” Type I IFNs: Type I IFNs in Intracellular Bacterial Infections
- Brucella Dysregulates Monocytes and Inhibits Macrophage Polarization through LC3-Dependent Autophagy
- Type I interferon signaling restrains IL-10R+ colonic macrophages and dendritic cells and leads to more severe Salmonella colitis
- Immunoproteasome Subunits Are Required for CD8+ T Cell Function and Host Resistance to Brucella abortus Infection in Mice
- Activation of macrophage mediated host defense against Salmonella typhimurium by Morus alba L.
Related papers
- Improving Pedagogy of Architecture with the Power of BIM
- Research on Teaching Design of Building Architecture Based on Bim Technology
- BIM-Enabled Pedagogy Approach: Using BIM as an Instructional Tool in Technology Courses
- Analysis of Curriculum Management and Proposal of BIM Educational Strategy in Architecture Design
- BIM in architecture curriculum: a case study
- Beyond BIM: Architecture Information Modeling