Slow Feedback Inhibition of Calcium Release-activated Calcium Current by Calcium Entry*
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Summary
The presence of a slow Ca2-dependent inactivation mechanism that curtails Ca2+ entry through CRAC channels is reported, likely to be an important factor that shapes the profile of theCa2+ signal in these nonexcitable cells.
- Type
- article
- Published
- 1998-06-12
- Cited by
- 104
- References
- 16
- Access
- Open access
- OpenAlex
- https://openalex.org/W9614097
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:26197863
Keywords
Plasticity, Materials science, Brittleness, Grain boundary, Slippage
References
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- Cell Type-specific Modes of Feedback Regulation of Capacitative Calcium Entry*
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- Depletion of intracellular calcium stores activates a calcium current in mast cells
- Depletion-activated calcium current is inhibited by protein kinase in RBL-2H3 cells.
- Thapsigargin, a tumor promoter, discharges intracellular Ca2+ stores by specific inhibition of the endoplasmic reticulum Ca2(+)-ATPase.
- Activation of store‐operated calcium influx at resting InsP3 levels by sensitization of the InsP3 receptor in rat basophilic leukaemia cells.
- A model for receptor-regulated calcium entry.
- An enzymatic mechanism for calcium current inactivation in dialysed Helix neurones.
- Rapid inactivation of depletion-activated calcium current (ICRAC) due to local calcium feedback
- The store-operated calcium current I(CRAC): nonlinear activation by InsP3 and dissociation from calcium release.
Cited by
- A possible cooperative structural transition of DNA in the 0.25-2.0 pN range.
- Calcium signalling and cancer cell growth.
- Molecular regulation of the pore component of CRAC channels, Orai1.
- Store-Operated Ca²⁺ Entry (SOCE) Pathways
- Store-operated calcium entry: mechanisms and modulation
- Discrete influx events refill depleted Ca2+ stores in a chick retinal neuron
- A Current Activated on Depletion of Intracellular Ca2+ Stores Can Regulate Exocytosis in Adrenal Chromaffin Cells
- Store-Operated Calcium Channels.
- The Wellcome Prize Lecture
- The calcium feedback loop and T cell activation: how cytoskeleton networks control intracellular calcium flux.
- Inwardly rectifying potassium currents in rat basophilic leukaemia (RBL-1) cells: regulation by spermine and implications for store-operated calcium influx
- The STIM1 CTID domain determines access of SARAF to SOAR to regulate Orai1 channel function
- Functional consequences of activating store-operated CRAC channels.
- Mitochondrial regulation of CRAC channel-driven cellular responses.
- Calmodulin regulates Ca(2+)-dependent feedback inhibition of store-operated Ca(2+) influx by interaction with a site in the C terminus of TrpC1.
- Polarity in intracellular calcium signaling.
- An examination of the secretion‐like coupling model for the activation of the Ca2+ release‐activated Ca2+ current ICRAC in RBL‐1 cells
- Store Operated Ca2+ Entry in Sensory Neurons: Functional Role and the Effect of Painful Nerve Injury
- Distinct Ca2+-permeable Cation Currents Are Activated by Internal Ca2+-Store Depletion in RBL-2H3 Cells and Human Salivary Gland Cells, HSG and HSY
- Suppression of human tumor cell proliferation through mitochondrial targeting
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