β1-adrenergic receptor stimulation by agonist Compound 49b restores insulin receptor signal transduction in vivo
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- Type
- article
- Published
- 2014-06-21
- Cited by
- 8
- References
- 21
- Access
- Open access
- OpenAlex
- https://openalex.org/W1853372
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:10260571
Keywords
Receptor, Agonist, Endocrinology, Internal medicine, Biology
References
- Application of Isoproterenol Inhibits Diabetic-Like Changes in the Rat Retina
- Diabetic retinopathy: more than meets the eye.
- Compound 49b protects against blast-induced retinal injury
- IRS-1-Mediated Inhibition of Insulin Receptor Tyrosine Kinase Activity in TNF-α- and Obesity-Induced Insulin Resistance
- Compound 49b prevents diabetes-induced apoptosis through increased IGFBP-3 levels.
- The role of TNFalpha and TNF receptors in obesity and insulin resistance.
- Increased basement membrane thickness, pericyte ghosts, and loss of retinal thickness and cells in dopamine beta hydroxylase knockout mice.
- β2-Adrenergic Receptor Knockout Mice Exhibit A Diabetic Retinopathy Phenotype
- Changes in expression of autonomic nerves in aging and disease.
- TNFα and SOCS3 regulate IRS-1 to increase retinal endothelial cell apoptosis
- SOCS-3 Inhibits Insulin Signaling and Is Up-regulated in Response to Tumor Necrosis Factor-α in the Adipose Tissue of Obese Mice*
- Role of beta-adrenergic receptors in inflammatory marker expression in Müller cells.
- Suppressor of Cytokine Signaling-3 (SOCS-3), a Potential Mediator of Interleukin-6-dependent Insulin Resistance in Hepatocytes*
- IGFBP-3 and TNF-α regulate retinal endothelial cell apoptosis.
- Insulin Signaling, Resistance, and the Metabolic Syndrome: Insights from Mouse Models to Disease Mechanisms
- Increased tumor necrosis factor-α, cleaved caspase 3 levels and insulin receptor substrate-1 phosphorylation in the β1-adrenergic receptor knockout mouse
- Silencing of insulin receptor substrate–1 increases cell death in retinal Müller cells
- β3-Adrenergic Receptors Regulate Retinal Endothelial Cell Migration and Proliferation*
Cited by
- Etanercept restores normal insulin signal transduction in β2-adrenergic receptor knockout mice
- β2–Adrenergic Receptor Antagonism Attenuates CNV Through Inhibition of VEGF and IL-6 Expression
- Inhibition of HMGB1 protects the retina from ischemia-reperfusion, as well as reduces insulin resistance proteins
- A regulatory role for β-adrenergic receptors regarding the resolvin D1 (RvD1) pathway in the diabetic retina
- miRNA15a regulates insulin signal transduction in the retinal vasculature
- Epac1 Restores Normal Insulin Signaling through a Reduction in Inflammatory Cytokines
- Inflammation In The Pathogenesis Of Diabetic Retinopathy
- Photoreceptor cells and RPE contribute to the development of diabetic retinopathy
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