Alzheimer's disease
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- Type
- review
- Published
- 2018-01-01
- Cited by
- 1,887
- References
- 77
- Access
- Open access
- OpenAlex
- https://openalex.org/W4235875658
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:24707616
Keywords
Medicine, Disease, Dementia, Epidemiology, Presentation (obstetrics)
References
- Neuroleptic drugs in dementia: benefits and harm
- Pathology of familial Alzheimer's disease with Lewy bodies.
- Uber eine eigenartige Erkrankung der Hirnrinde
- Preclinical trials in autosomal dominant AD: Implementation of the DIAN-TU trial
- A Critique of the Drug Discovery and Phase 3 Clinical Programs Targeting the Amyloid Hypothesis for Alzheimer Disease
- Nursing home placement in the Donepezil and Memantine in Moderate to Severe Alzheimer's Disease (DOMINO-AD) trial: secondary and post-hoc analyses.
- Review: An update on clinical, genetic and pathological aspects of frontotemporal lobar degenerations
- Meta-analysis of modifiable risk factors for Alzheimer's disease
- Dementia in Western Europe: epidemiological evidence and implications for policy making
- Phases of Aβ-deposition in the human brain and its relevance for the development of AD
- Early Aβ accumulation and progressive synaptic loss, gliosis, and tangle formation in AD brain
- Alzheimer's disease risk genes and mechanisms of disease pathogenesis
- Alzheimer's disease and other neurological disorders
- Correlating familial Alzheimer's disease gene mutations with clinical phenotype
- Cholinesterase Inhibitors in Alzheimer's Disease
- Self-propagation of pathogenic protein aggregates in neurodegenerative diseases
- The clinical use of structural MRI in Alzheimer disease
- Neuropathological stageing of Alzheimer-related changes
- Roles of Apolipoprotein E in Alzheimer’s Disease and Other Neurological Disorders
- EFNS‐ENS/EAN Guideline on concomitant use of cholinesterase inhibitors and memantine in moderate to severe Alzheimer's disease
Cited by
- Umbilical Cord Mesenchymal Stem Cells Conditioned Medium Promotes Aβ25-35 phagocytosis by Modulating Autophagy and Aβ-Degrading Enzymes in BV2 Cells
- Role of epigenetics in Alzheimer's disease pathogenesis.
- Resveratrol Induces Brain Resilience Against Alzheimer Neurodegeneration Through Proteostasis Enhancement
- Immunotherapy to improve cognition and reduce pathological species in an Alzheimer’s disease mouse model
- Mutations of Histidine 13 to Arginine and Arginine 5 to Glycine Are Responsible for Different Coordination Sites of Zinc(II) to Human and Murine Peptides.
- Recent advancements toward therapeutic vaccines against Alzheimer’s disease
- A Comparison of Lifestyle Interventions for Alzheimer's Disease Extracted from Clinical Notes and Literature
- Omics-based biomarkers for the early Alzheimer Disease diagnosis and reliable therapeutic targets development.
- A cross-species approach to disorders affecting brain and behaviour
- Visual Features in Alzheimer's Disease: From Basic Mechanisms to Clinical Overview
- The protective effects of polyphenols on Alzheimer's disease: A systematic review
- Liraglutide and a lipidized analog of prolactin‐releasing peptide show neuroprotective effects in a mouse model of &bgr;‐amyloid pathology
- MicroRNA‐128 knockout inhibits the development of Alzheimer's disease by targeting PPAR&ggr; in mouse models
- New approaches for the treatment of Alzheimer's disease.
- Rare genetic variation implicated in non-Hispanic white families with Alzheimer disease
- The Challenge to Search for New Nervous System Disease Biomarker Candidates: the Opportunity to Use the Proteogenomics Approach
- Expansion of the scaffold diversity for the development of highly selective butyrylcholinesterase (BChE) inhibitors: Discovery of new hits through the pharmacophore model generation, virtual screening and molecular dynamics simulation.
- Amyloid, tau, pathogen infection and antimicrobial protection in Alzheimer’s disease –conformist, nonconformist, and realistic prospects for AD pathogenesis
- Sitagliptin protects the cognition function of the Alzheimer's disease mice through activating glucagon-like peptide-1 and BDNF-TrkB signalings.
- Novel, non-nitrocatechol COMT inhibitors modulate dopamine neurotransmission in the frontal cortex and improve cognitive flexibility
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