Control of transcription activation by steroid hormone receptors
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Summary
S steroid hormone‐regulated gene transcription involves a multitude of interactive elements, as is expected from the central role of nuclear receptors in homeostasis, embryonic development, and differentiation.
- Type
- review
- Published
- 1992-05-01
- Cited by
- 212
- References
- 68
- OpenAlex
- https://openalex.org/W2121317812
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:24380709
Keywords
Hormone, Receptor, Steroid hormone, Steroid, Transcription (linguistics)
References
- Signal transduction by steroid hormones: nuclear localization is differentially regulated in estrogen and glucocorticoid receptors.
- Distinct classes of transcriptional activating domains function by different mechanisms.
- Solution structure of the DNA-binding domain of the oestrogen receptor.
- Determinants of target gene specificity for steroid/thyroid hormone receptors.
- The estrogen‐responsive element as an inducible enhancer: DNA sequence requirements and conversion to a glucocorticoid‐responsive element.
- Cooperative binding of estrogen receptor to imperfect estrogen‐responsive DNA elements correlates with their synergistic hormone‐dependent enhancer activity.
- The transcriptional activation function located in the hormone‐binding domain of the human oestrogen receptor is not encoded in a single exon.
- Mutual transrepression of Fos and the glucocorticoid receptor: involvement of a functional domain in Fos which is absent in FosB.
- Cell‐specific inhibitory and stimulatory effects of Fos and Jun on transcription activation by nuclear receptors.
- Glucocorticoid receptor binds cooperatively to adjacent recognition sites.
- Two signals mediate hormone‐dependent nuclear localization of the glucocorticoid receptor.
- In contrast to the glucocorticoid receptor, the thyroid hormone receptor is translated in the DNA binding state and is not associated with hsp90.
- Transcriptional control by nuclear receptors
- The cloned human oestrogen receptor contains a mutation which alters its hormone binding properties.
- Two distinct estrogen‐regulated promoters generate transcripts encoding the two functionally different human progesterone receptor forms A and B.
- Role of the two activating domains of the oestrogen receptor in the cell‐type and promoter‐context dependent agonistic activity of the anti‐oestrogen 4‐hydroxytamoxifen.
- A common ancestor DNA motif for invertebrate and vertebrate hormone response elements.
- Superfamily of steroid nuclear receptors: positive and negative regulators of gene expression
- A retinoic acid response element is present in the mouse cellular retinol binding protein I (mCRBPI) promoter.
- The chicken progesterone receptor: sequence, expression and functional analysis.
Cited by
- Nuclear remodeling in response to steroid hormone action.
- Molecular determinants for agonist and antagonist binding to steroid nuclear receptors.
- Thymocyte apoptosis by glucocorticoids and cAMP.
- Effect of Oral Contraceptives on Haemostasis Variables
- Signalling Mechanisms — from Transcription Factors to Oxidative Stress
- Regulation of peroxisome proliferator-activated receptors.
- Steroid synthesis and metabolism in the nervous system: Trophic and protective effects
- Genetic analysis of androgen receptors in development and disease.
- Antiestrogenic effects of all-trans-retinoic acid and 1,25-dihydroxyvitamin D3 in breast cancer cells occur at the estrogen response element level but through different molecular mechanisms.
- Acceleration in the Rate of CNS Remyelination in Lysolecithin-Induced Demyelination
- Anti-inflammatory actions of glucocorticoids: molecular mechanisms.
- cAMP-dependent regulation of gene transcription by cAMP response element-binding protein and cAMP response element modulator.
- Suppression of sodium channel function in differentiating C2 muscle cells stably overexpressing rat androgen receptors
- Differential regulation of peptide alpha-amidation by dexamethasone and disulfiram.
- Suppression of Granulocyte-Macrophage Colony-Stimulating Factor Expression by Glucocorticoids Involves Inhibition of Enhancer Function by the Glucocorticoid Receptor Binding to Composite NF-AT/Activator Protein-1 Elements1
- Dysfunctional glucocorticoid receptor with a single point mutation ablates the CCAAT/enhancer binding protein‐dependent growth suppression response in a steroid‐resistant rat hepatoma cell variant
- Therapeutic Benefit of a Dissociated Glucocorticoid and the Relevance of In Vitro Separation of Transrepression from Transactivation Activity
- In vitro and in vivo interactions between nuclear receptors at estrogen response elements.
- TAXI/UAS: A Molecular Switch to Control Expression of Genes In Vivo
- Human vascular smooth muscle cells contain functional estrogen receptor.
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