A mouse model of autoimmune encephalitis.
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Summary
This scientific commentary refers to ‘Human N-methyl D-aspartate receptor antibodies alter memory and behaviour in mice’ by Planaguma et al (doi: 10.1093/brain/awu310).
- Type
- letter
- Published
- 2015-01-01
- Cited by
- 2
- References
- 14
- Access
- Open access
- OpenAlex
- https://openalex.org/W2030047612
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:6033458
Keywords
Autoimmune encephalitis, Encephalitis, Neuroscience, Antibody, Virology
References
- In vivo effects of antibodies from patients with anti-NMDA receptor encephalitis: further evidence of synaptic glutamatergic dysfunction
- Acute Mechanisms Underlying Antibody Effects in Anti–N-Methyl-D-Aspartate Receptor Encephalitis
- Suppression of synaptic plasticity by cerebrospinal fluid from anti-NMDA receptor encephalitis patients.
- Extrasynaptic NMDA receptor involvement in central nervous system disorders.
- NMDA Neurotransmission Dysfunction in Behavioral and Psychological Symptoms of Alzheimer’s Disease
- NMDA receptor subunit diversity: impact on receptor properties, synaptic plasticity and disease
- Reversing EphB2 depletion rescues cognitive functions in Alzheimer model
- Cellular and synaptic mechanisms of anti-NMDA receptor encephalitis
- Human N-methyl D-aspartate receptor antibodies alter memory and behaviour in mice.
- Disrupted surface cross-talk between NMDA and Ephrin-B2 receptors in anti-NMDA encephalitis.
- Inducible and reversible NR1 knockout reveals crucial role of the NMDA receptor in preserving remote memories in the brain.
- Surface dynamics of GluN2B‐NMDA receptors controls plasticity of maturing glutamate synapses
- Treatment and prognostic factors for long-term outcome in patients with anti-N-Methyl-D-Aspartate (NMDA) receptor encephalitis: a cohort study
- Activity-dependent dendritic spine neck changes are correlated with synaptic strength
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