In vivo effects of antibodies from patients with anti-NMDA receptor encephalitis: further evidence of synaptic glutamatergic dysfunction

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Summary

Evidence in vivo is provided that NMDA-R antibodies deregulate the glutamatergic pathways and that the encephalitis associated with these antibodies is an auto-immune synaptic disorder.

Type
article
Published
2010-11-26
Cited by
138
References
28
Access
Open access

Keywords

Computer science

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