Tumor dormancy and cell signaling: anti-mu-induced apoptosis in human B-lymphoma cells is not caused by an APO-1-APO-1 ligand interaction.
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Summary
It is shown that crosslinking the membrane immunoglobulin on human lymphoma cells (Daudi) does not induce synthesis of APO-1 ligand, and in B-lymphoma cells, apoptosis induced by signaling via membrane IgM is not mediated by the APO the ligand.
- Type
- article
- Published
- 1996-03-05
- Cited by
- 21
- References
- 0
- Access
- Open access
- OpenAlex
- https://openalex.org/W2004124992
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:23782345
Keywords
Apoptosis, Lymphoma, Signal transduction, Cell biology, Biology
References
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Cited by
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- Dormant tumor cells as a therapeutic target?
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- Tumor dormancy and immunoescape
- Autocrine cell suicide in a Burkitt lymphoma cell line (Daudi) induced by interferon alpha: involvement of tumor necrosis factor as ligand for the CD95 receptor.
- Natural Anti-Immunoglobulin Autoantibodies: Irrelevant By-Products or Immunoregulatory Molecules?
- Dextran sulfate-induced degradation of spontaneously apoptotic B cells.
- Pivotal Advance: CEACAM1 is a negative coreceptor for the B cell receptor and promotes CD19‐mediated adhesion of B cells in a PI3K‐dependent manner
- Apoptosis as a Scaffold for Building up the B Cell Repertoire
- DNA vaccination against cancer antigens.
- The current paradigm and challenges ahead for the dormancy of disseminated tumor cells
- Prevention of Anti-IgM-Induced Apoptosis Accompanying G 1 Arrest in B Lymphoma Cells Overexpressing Dominant-Negative Mutant Form of c-Jun
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