Association of neurotrophin receptor expression and differentiation in human neuroblastoma.
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Summary
The existence and spatial distribution of neurotrophin receptors in neuroblastoma lend supportive evidence that neurotrophic influences may be involved in tumor persistence or regression.
- Type
- article
- Published
- 1995-07-01
- Cited by
- 94
- References
- 39
- Access
- Open access
- OpenAlex
- https://openalex.org/W1567431652
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:32011768
Keywords
Tropomyosin receptor kinase A, Neurotrophin, Tropomyosin receptor kinase C, Tropomyosin receptor kinase B, Biology
References
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- Coexpression of messenger RNA for TRK protooncogene and low affinity nerve growth factor receptor in neuroblastoma with favorable prognosis.
- Regulation of neurotrophin receptors during the maturation of the mammalian visual system
- Expression and function of TRK-B and BDNF in human neuroblastomas
- Neurotrophins and their receptors--current concepts and implications for neurologic disease.
- Lack of high-affinity nerve growth factor receptors in aggressive neuroblastomas.
- trk and ret proto‐oncogene expression in human neuroblastoma specimens: High frequency of trk expression in non‐advanced stages
- Neuronal colocalization of mRNAs for neurotrophins and their receptors in the developing central nervous system suggests a potential for autocrine interactions.
- A proposed staging for children with neuroblastoma. Children's cancer study group A
- Toward an understanding of the molecular mechanisms of physiological cell death.
- Tissue-specific expression of the human tropomyosin gene involved in the generation of the trk oncogene
- Trk receptors use redundant signal transduction pathways involving SHC and PLC-γ1 to mediate NGF responses
- The nerve growth factor 35 years later.
- Induction of TrkB by retinoic acid mediates biologic responsiveness to BDNF and differentiation of human neuroblastoma cells. Eukaryotic Signal Transduction Group.
- Expression of members of the trk family in the developing postnatal rat brain.
- Keeping track of neurotrophin receptors.
- Targeted disruption of the trkB neurotrophin receptor gene results in nervous system lesions and neonatal death.
Cited by
- Neuroblastoma as an experimental model for neuronal differentiation and hypoxia-induced tumor cell dedifferentiation.
- Expression of nerve growth factor, p75, and the high affinity neurotrophin receptors in the adult rat trigeminal system: Evidence for multiple trophic support systems
- Mechanisms of neuroblastoma regression
- Expression of neurotrophins and their receptors in peripheral lung cells of mice
- Expression of brain-derived neurotrophic factor and tyrosine kinase B receptor proteins in glioneuronal tumors from patients with intractable epilepsy: colocalization with N-methyl-D-aspartic acid receptor
- Neurotrophin and neurotrophin receptor proteins in medulloblastomas and other primitive neuroectodermal tumors of the pediatric central nervous system.
- Molecular mechanisms regulating embryonic cerebral cortex development.
- Neurotrophin Receptors TrkA and TrkB in Retinoblastoma Are Differentially Expressed Depending on Cellular Differentiation
- Trophic factors in the carotid body.
- In vivo spontaneous neuronal to neuroendocrine lineage conversion in a subset of neuroblastomas.
- Cellular localization of the Trk neurotrophin receptor family in human non-neuronal tissues.
- Hypoxia and differentiation in human neuroblastoma cells
- Differences in early and late responses between neurotrophin-stimulated trkA- and trkC-transfected SH-SY5Y neuroblastoma cells.
- Oncogenic Pathways and Molecular Prognostics in Neuroblastoma
- Transcriptional Regulation by Hypoxia-Inducible Factors in Tumor Cells
- Hypoxia-induced phenotypic modulation of human neuroblastoma cells
- Cathepsin D is a marker of ganglion cell differentiation in the developing and neoplastic human peripheral sympathetic nervous tissues
- Neuroblastoma aggressiveness in relation to sympathetic neuronal differentiation stage.
- TrkA Induces Apoptosis of Neuroblastoma Cells and Does So via a p53-dependent Mechanism*[boxs]
- ΔNp73 Modulates Nerve Growth Factor-Mediated Neuronal Differentiation through Repression of TrkA
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