Epigenetics and targeting mechanisms in Drosophila melanogaster
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Summary
It is proposed that the role of roX RNAs is to prevent the binding of the MSL-complex to heterochromatin and the sequence analysis showed that in the absence of ro X RNAs, the MSl-complex has an affinity for regions enriched in Hoppel transposable elements and repeats in general.
- Type
- article
- Published
- 2015-01-01
- Cited by
- 0
- References
- 223
- Access
- Open access
- OpenAlex
- https://openalex.org/W281563292
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:82902859
Keywords
Dosage compensation, Biology, Heterochromatin, Drosophila melanogaster, Polytene chromosome
References
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- Sex Chromosomes and Sex-Linked Genes
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- Selective recognition of methylated lysine 9 on histone H3 by the HP1 chromo domain
- Nucleosome distribution and linker DNA: connecting nuclear function to dynamic chromatin structure
- The Drosophila SET domain encoding gene dEset is essential for proper development.
- Cellular mechanism for targeting heterochromatin formation in Drosophila.
- Heterochromatin protein 1 binds to nucleosomes and DNA in vitro.
- Methylation of histone H3 lysine 9 creates a binding site for HP1 proteins
- Male-Specific Lethal Mutations of DROSOPHILA MELANOGASTER . II. Parameters of Gene Action during Male Development.
- The heterochromatin-associated protein HP-1 is an essential protein in Drosophila with dosage-dependent effects on position-effect variegation.
- Stepwise assembly of chromatin during DNA replication in vitro.
- The effects of chromosome rearrangements on the expression of heterochromatic genes in chromosome 2L of Drosophila melanogaster.
- Drosophila male-specific lethal-2 protein: structure/function analysis and dependence on MSL-1 for chromosome association.
- The Drosophila roX1 RNA gene can overcome silent chromatin by recruiting the male-specific lethal dosage compensation complex.
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