Herpes Simplex Virus and Interferon Signaling Induce Novel Autophagic Clusters in Sensory Neurons
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Summary
Results show that infection with HSV and antiviral signaling in TG neurons produce an unorthodox autophagic response that may represent an important neuronal response to HSV infection and the establishment of latency, and may have important implications for the understanding of latency and reactivation.
- Type
- article
- Published
- 2016-02-24
- Cited by
- 46
- References
- 92
- OpenAlex
- https://openalex.org/W26912623
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:4767267
Keywords
Quickening, Electrification, Work (physics), Decree, Hydropower
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- Rapid Etiologic Diagnosis of Herpes Virus Infections in Children
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- ISGylation – a key to lock the cell gates for preventing the spread of threats
- Isolation, Purification, and Culture of Primary Murine Sensory Neurons
- Pegylated interferon beta in the treatment of the Theiler's murine encephalomyelitis virus mouse model of multiple sclerosis.
- Herpesvirus and Autophagy: “All Right, Everybody Be Cool, This Is a Robbery!”
- Transmembrane Protein pUL50 of Human Cytomegalovirus Inhibits ISGylation by Downregulating UBE1L
- Customized autophagy: a long way to go
- Interplay Between the Autophagy-Lysosomal Pathway and the Ubiquitin-Proteasome System: A Target for Therapeutic Development in Alzheimer’s Disease
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- Programmed cell death: the battlefield between the host and alpha-herpesviruses and a potential avenue for cancer treatment
- Insights into the pathogenesis of herpes simplex encephalitis from mouse models
- ISG15 in cancer: Beyond ubiquitin-like protein.
- Human TANK-binding kinase 1 is required for early autophagy induction upon herpes simplex virus 1 infection.
- Autophagy-Virus Interplay: From Cell Biology to Human Disease
- The ESCRT-Related ATPase Vps4 Is Modulated by Interferon during Herpes Simplex Virus 1 Infection
- Disturbed Yin–Yang balance: stress increases the susceptibility to primary and recurrent infections of herpes simplex virus type 1