Reactive Oxygen Species and Targeted Therapy for Pancreatic Cancer
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Summary
Reactive oxygen species (ROS) plays the vital roles in the progression of pancreatic cancer and in the response to cancer treatment and may be the novel target for various strategies and drugs to modulate ROS levels in pancreaticcancer therapy.
- Type
- dissertation
- Published
- 2016-01-03
- Cited by
- 110
- References
- 102
- Access
- Open access
- OpenAlex
- https://openalex.org/W26881012
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:18180828
Keywords
Foreign direct investment, Intellectual property, Business, Law and economics, Monetary economics
References
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- Mitochondrial ROS in cancer: initiators, amplifiers or an Achilles’ heel?
- Inhibition of cell proliferation and induction of apoptosis by CDDO-Me in pancreatic cancer cells is ROS-dependent
- Tumorigenesis: Oncogene detox programme
- Anti-cancer effect of resveratrol is associated with induction of apoptosis via a mitochondrial pathway alignment.
- The measurement of glutathione in human epidermis using glutathione reductase.
- Reactive oxygen species signaling in cancer: comparison with aging.
- The role of manganese superoxide dismutase in the growth of pancreatic adenocarcinoma.
- Non-invasive radiofrequency treatment effect on mitochondria in pancreatic cancer cells
- JNK suppression of chemotherapeutic agents-induced ROS confers chemoresistance on pancreatic cancer stem cells
- STAT3 down regulates LC3 to inhibit autophagy and pancreatic cancer cell growth
- PLGA-Loaded Gold-Nanoparticles Precipitated with Quercetin Downregulate HDAC-Akt Activities Controlling Proliferation and Activate p53-ROS Crosstalk to Induce Apoptosis in Hepatocarcinoma Cells
- Oleanolic acid arrests cell cycle and induces apoptosis via ROS‐mediated mitochondrial depolarization and lysosomal membrane permeabilization in human pancreatic cancer cells
- Onconase induces autophagy sensitizing pancreatic cancer cells to gemcitabine and activates Akt/mTOR pathway in a ROS-dependent manner.
- Modulation of oxidative stress as an anticancer strategy
- Superoxide Anion Radical (O·̄2), Superoxide Dismutases, and Related Matters*
- Longikaurin E induces apoptosis of pancreatic cancer cells via modulation of the p38 and PI3K/AKT pathways by ROS
Cited by
- CD38 enhances the proliferation and inhibits the apoptosis of cervical cancer cells by affecting the mitochondria functions
- Reactive Oxygen Species Regulate T Cell Immune Response in the Tumor Microenvironment
- Metals and metastasis: Exploiting the role of metals in cancer metastasis to develop novel anti‐metastatic agents
- MicroRNA-497 regulates cisplatin chemosensitivity of cervical cancer by targeting transketolase.
- Pancreatic Cancer: Historical Problem as well as Personal and Current Solutions which have Prospects in Translational Medicine
- Induction of Oxidants Distinguishes Susceptibility of Prostate Carcinoma Cell Lines to p53 Gene Transfer Mediated by an Improved Adenoviral Vector
- Targeting a Common Enemy: Toxic Cellular Mechanism of Novel Anti-cancer Agents that Alter DNA and Transcription
- Oxysterol-Binding Protein-Related Protein 8 Inhibits Gastric Cancer Growth Through Induction of ER Stress, Inhibition of Wnt Signaling, and Activation of Apoptosis
- A novel deubiquitinase inhibitor b-AP15 triggers apoptosis in both androgen receptor-dependent and -independent prostate cancers
- Superoxide Dismutases in Pancreatic Cancer
- The receptor for advanced glycation end products: A fuel to pancreatic cancer.
- Monascus purpureus induced apoptosis on gastric cancer cell by scavenging mitochondrial reactive oxygen species
- Synthesis of 2-Substituted Benzothiazole Derivatives and Their In Vitro Anticancer Effects and Antioxidant Activities Against Pancreatic Cancer Cells.
- An overview of polyamine metabolism in pancreatic ductal adenocarcinoma
- Cancer cell lines involving cancer stem cell populations respond to oxidative stress
- Disorders noticed during development of pancreatic cancer: potential opportunities for early and effective diagnostics and therapy
- Nanotherapy and Reactive Oxygen Species (ROS) in Cancer: A Novel Perspective
- Mitigating the Impact of Antibacterial Drug Resistance through Host-Directed Therapies: Current Progress, Outlook, and Challenges
- The role of oxidative stress in activity of anticancer thiosemicarbazones
- p53 and metabolism: from mechanism to therapeutics
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