Characterization of miR-888 expression and regulation in endometrial cancer
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Summary
It is concluded that miR-888 expression marks an aggressive endometrial tumor phenotype, which is a potent tumor suppressor of the endometrium whose expression is often lost in advancedendometrial cancers.
- Type
- dissertation
- Published
- 2014-05-01
- Cited by
- 0
- References
- 136
- Access
- Open access
- OpenAlex
- https://openalex.org/W25255620
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:81562694
Keywords
Endometrial cancer, microRNA, Cancer, Cancer research, Serous fluid
References
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- Integrated Genomic Characterization of Endometrial Carcinoma
- The Expression and Ovarian Steroid Regulation of Endometrial Micro-RNAs
- Two distinct estrogen‐regulated promoters generate transcripts encoding the two functionally different human progesterone receptor forms A and B.
- p53 gene mutations are common in uterine serous carcinoma and occur early in their pathogenesis.
- Oncomirs — microRNAs with a role in cancer
- Progesterone receptor B gene inactivation and CpG hypermethylation in human uterine endometrial cancer.
- Hormonal contraception for human males: prospects.
- Exosome: from internal vesicle of the multivesicular body to intercellular signaling device.
- The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2.
- Carcinosarcomas (malignant mixed Müllerian tumor) of the uterus: advances in elucidation of biologic and clinical characteristics.
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