TIM-1 deficiency eliminates airway hyperreactivity triggered by the recognition of airway cell death

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Summary

A novel pathway in which TIM-1, a receptor for phosphatidylserine expressed by apoptotic cells, drives the development of asthma by sensing and responding to injured and apoptotic airway epithelial cells is defined.

Type
article
Published
2013-05-11
Cited by
30
References
57
Access
Open access

Keywords

Cobalt, Kinetics, Hydrogen, Chemistry, Deuterium

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