Working hard for recovery: mitotic kinases in the DNA damage checkpoint
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Summary
The emerging role of mitotic kinases in the DNA damage pathway provides important insights into cancer progression and treatment.
- Type
- article
- Published
- 2013-04-23
- Cited by
- 11
- References
- 71
- Access
- Open access
- OpenAlex
- https://openalex.org/W23618492
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:14357303
Keywords
Globalization, Political science, Sociology, Philosophy, Art history
References
- Checkpoint recovery after DNA damage: a rolling stop for CDKs
- Enforcing the Greatwall in Mitosis
- DNA double-strand breaks: signaling, repair and the cancer connection
- Polo-like kinase-1 is a target of the DNA damage checkpoint
- Aurora-A — A guardian of poles
- Cell Cycle-dependent Complex Formation of BRCA1·CtIP·MRN Is Important for DNA Double-strand Break Repair*
- The DNA damage response: putting checkpoints in perspective
- Abrogation of the Chk1-mediated G(2) checkpoint pathway potentiates temozolomide-induced toxicity in a p53-independent manner in human glioblastoma cells.
- Cdk-mediated phosphorylation of Chk1 is required for efficient activation and full checkpoint proficiency in response to DNA damage
- Polo-like kinase 1 reaches beyond mitosis--cytokinesis, DNA damage response, and development.
- Human CtIP Mediates Cell Cycle Control of DNA End Resection and Double Strand Break Repair
- The CDK regulates repair of double‐strand breaks by homologous recombination during the cell cycle
- Human CtIP promotes DNA end resection
- Control of the G2/M checkpoints after exposure to low doses of ionising radiation: implications for hyper-radiosensitivity.
- Behind the wheel and under the hood: functions of cyclin-dependent kinases in response to DNA damage
- DNA damage checkpoints: from initiation to recovery or adaptation.
- DNA Damage Response Pathways in Tumor Suppression and Cancer Treatment
- DNA end resection, homologous recombination and DNA damage checkpoint activation require CDK1
- Cell cycle regulation of DNA double-strand break end resection by Cdk1-dependent Dna2 phosphorylation
- Cell Cycle-Dependent Regulation of Double-Strand Break Repair: A Role for the CDK
Cited by
- Effects of Turkish propolis on expression of hOGG-1 and NEIL-1.
- Fanconi anemia cells with unrepaired DNA damage activate components of the checkpoint recovery process
- Actual Isothermal Effects of Water‐Filtered Infrared A‐Irradiation
- Cell fate determination in cisplatin resistance and chemosensitization
- HOXC10 expression supports the development of chemotherapy resistance by fine tuning DNA repair in breast cancer cells
- THE HOMEOBOX PROTEIN HOXC10 IS OVEREXPRESSED IN BREAST CANCER AND INCREASES RESISTANCE TO CHEMOTHERAPY
- WIP1 Contributes to the Adaptation of Fanconi Anemia Cells to DNA Damage as Determined by the Regulatory Network of the Fanconi Anemia and Checkpoint Recovery Pathways
- Comparative transcriptome and proteome provide new insights into the regulatory mechanisms of the postharvest deterioration of Pleurotus tuoliensis fruitbodies during storage.
- An In Vitro Study of Saffron Carotenoids: The Effect of Crocin Extracts and Dimethylcrocetin on Cancer Cell Lines
- DNA damage checkpoint execution and the rules of its disengagement
- The ATM-E6AP-MASTL axis mediates DNA damage checkpoint recovery
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