Trichinella spiralis: nurse cell formation with emphasis on analogy to muscle cell repair
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Summary
Some events that occur during the nurse cell formation are analogous to those occurring during muscle cell regeneration/repair during Trichinella infection.
- Type
- article
- Published
- 2008-08-19
- Cited by
- 114
- References
- 133
- Access
- Open access
- OpenAlex
- https://openalex.org/W2089406333
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:14752050
Keywords
Biology, Nurse cell, Trichinella spiralis, Cell, Cell biology
References
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- How does Trichinella spiralis make itself at home?
- MRF4 protein expression in regenerating rat muscle
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- Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways
- Origin of the blastema in regenerating limbs of the newt Triturus viridescens. An autoradiographic study using tritiated thymidine to follow cell proliferation and migration.
- Localized Igf-1 transgene expression sustains hypertrophy and regeneration in senescent skeletal muscle
- Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo
- Manipulating the onset of cell cycle withdrawal in differentiated erythroid cells with cyclin-dependent kinases and inhibitors.
- Analysis of a 43-kDa glycoprotein from the intracellular parasitic nematode Trichinella spiralis.
- Progression through the G1‐phase of the on‐going cell cycle
- The G0/G1 switch gene 2 is a novel PPAR target gene.
- Cyclin D-dependent kinases, INK4 inhibitors and cancer.
- MyoD and the transcriptional control of myogenesis.
- Electron microscopic observations of muscle dedifferentiation in regenerating amblystoma limbs
- Crosstalk between cell cycle regulators and the myogenic factor MyoD in skeletal myoblasts
- Mitochondria as the central control point of apoptosis.
- Clusterin and DNA repair: a new function in cancer for a key player in apoptosis and cell cycle control
Cited by
- FOXO1, TGF-β Regulation and Wound Healing
- Necrosis and apoptosis in Trichinella spiralis-mediated tumour reduction
- Secretory Products of Trichinella spiralis Muscle Larvae and Immunomodulation: Implication for Autoimmune Diseases, Allergies, and Malignancies
- Mechanisms of modulation of experimental autoimmune encephalomyelitis by chronic Trichinella spiralis infection in Dark Agouti rats
- Trichinella spiralis: shaping the immune response
- The impact of Trichinella spiralis excretory-secretory products on dendritic cells.
- The Importance of the Nurse Cells and Regulatory Cells in the Control of T Lymphocyte Responses
- Trichinella: differential expression of angiogenic factors in macrophages stimulated with antigens from encapsulated and non-encapsulated species.
- Trichinella spiralis shares epitopes with human autoantigens.
- Apoptosis as the adaptation mechanism in survival of Trichinella spiralis in the host
- Application of dendritic cells stimulated with Trichinella spiralis excretory–secretory antigens alleviates experimental autoimmune encephalomyelitis
- Trichinella spiralis infection induces angiogenic factor thymosin β4 expression.
- Therapeutic Potential of Myrrh and Ivermectin against Experimental Trichinella spiralis Infection in Mice
- Inhibition of mammalian muscle differentiation by excretory secretory products of muscle larvae of Trichinella spiralis in vitro
- Trichinella: what is going on during nurse cell formation?
- Mapping of the complement C9 binding domain on Trichinella spiralis paramyosin
- Temporal quantification of Ts43 gene expression of Trichinella spiralis using real-time RT-qPCR.
- An anti-tumor protein produced by Trichinella spiralis induces apoptosis in human hepatoma H7402 cells.
- Parasitic infections and myositis
- Regulation of cytokine expression in murine macrophages stimulated by excretory/secretory products from Trichinella spiralis in vitro
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