Regression of Dalton’s lymphoma in vivo via decline in lactate dehydrogenase and induction of apoptosis by a ruthenium(II)-complex containing 4-carboxy N-ethylbenzamide as ligand

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Summary

Modulation of M4-LDH by this compound in a T-cell lymphoma (Dalton’s Lymphoma: DL) vis a vis regression of the tumor in vivo is described to suggest that Ru(II)-CNEB is able to regress Dalton’'s lymphoma in vivo via declining M4 -LDH and inducing mitochondrial dysfunction–apoptosis pathway without producing any toxicity to the normal tissues.

Type
article
Published
2009-12-01
Cited by
38
References
52

Keywords

Lactate dehydrogenase, In vivo, Apoptosis, Cytotoxicity, DNA fragmentation

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