Persistent infection of SARS coronavirus in colonic cells in vitro
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Summary
The results on the expression of angiotensin‐converting enzyme 2 (ACE2), a recently identified cellular receptor for SARS‐CoV, in these cell lines indicated that it might not be the sole determinant for cells to be susceptible to SARS-CoV infection.
- Type
- article
- Published
- 2004-07-13
- Cited by
- 95
- References
- 24
- Access
- Open access
- OpenAlex
- https://openalex.org/W2056296106
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:38988727
Keywords
Vero cell, Virology, Cytopathic effect, Coronaviridae, Biology
References
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- Proliferative growth of SARS coronavirus in Vero E6 cells.
- Pulmonary pathological features in coronavirus associated severe acute respiratory syndrome (SARS)
- Severe Acute Respiratory Syndrome–associated Coronavirus Infection
- Clinical presentations and outcome of severe acute respiratory syndrome in children
- Immunofluorescence Assay for Serologic Diagnosis of SARS
- A 193-Amino Acid Fragment of the SARS Coronavirus S Protein Efficiently Binds Angiotensin-converting Enzyme 2
Cited by
- Severe Acute Respiratory Syndrome and Coronavirus
- Insights from the Association of SARS-CoV S-Protein with its Receptor, ACE2
- Severe Acute Respiratory Syndrome Coronavirus Entry as a Target of Antiviral Therapies
- MOLECULAR AND GENOMIC APPROACHES TO UNDERSTANDING HOST-VIRUS INTERACTIONS IN SHAPING THE OUTCOME OF EQUINE ARTERITIS VIRUS INFECTION
- Studies on human pathogenic coronaviruses survey for coronaviruses in African bat species and characterization of the novel human coronavirus NL63 (HCoV-NL63) open reading frame 3 (ORF3)
- JNK and PI3k/Akt signaling pathways are required for establishing persistent SARS-CoV infection in Vero E6 cells
- Quantitative temporal‐spatial distribution of severe acute respiratory syndrome‐associated coronavirus (SARS‐CoV) in post‐mortem tissues
- Pathology and Pathogenesis of Severe Acute Respiratory Syndrome
- Bilateral Entry and Release of Middle East Respiratory Syndrome Coronavirus Induces Profound Apoptosis of Human Bronchial Epithelial Cells
- Exogenous ACE2 Expression Allows Refractory Cell Lines To Support Severe Acute Respiratory Syndrome Coronavirus Replication
- Microarray and real-time RT-PCR analyses of differential human gene expression patterns induced by severe acute respiratory syndrome (SARS) coronavirus infection of Vero cells
- Cell Type-Specific Cleavage of Nucleocapsid Protein by Effector Caspases during SARS Coronavirus Infection
- How the SARS coronavirus causes disease: host or organism?
- SARS: clinical presentation, transmission, pathogenesis and treatment options.
- Enhancement of cytotoxicity against Vero E6 cells persistently infected with SARS-CoV by Mycoplasma fermentans
- Mechanisms of establishment of persistent SARS-CoV-infected cells
- Apical Entry and Release of Severe Acute Respiratory Syndrome-Associated Coronavirus in Polarized Calu-3 Lung Epithelial Cells
- SARS-CoV accessory protein 7a directly interacts with human LFA-1
- Establishment of Vero E6 cell clones persistently infected with severe acute respiratory syndrome coronavirus
- Susceptibility of different eukaryotic cell lines to SARS-coronavirus
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