Prothymosin α overexpression contributes to the development of pulmonary emphysema

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Summary

It is shown that prothymosin α contributes to the pathogenesis of emphysema by increasing acetylation of histones and nuclear factor-kappaB, particularly upon cigarette smoke exposure, and that exposure to cigarette smoke increases prothyrosin α expression.

Type
article
Published
2013-05-21
Cited by
38
References
68
Access
Open access

Keywords

Pathogenesis, Matrix metalloproteinase, Medicine, Pathology, Cancer research

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