A permanent change in brain function resulting from daily electrical stimulation.
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Summary
High-intensity stimulation studies revealed that the development of convulsions was not based simply on threshold reduction, but involved complex reorganization of function.
- Type
- article
- Published
- 1969-11-01
- Cited by
- 3,007
- References
- 14
- OpenAlex
- https://openalex.org/W2019884656
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:44288404
Keywords
Stimulation, Convulsion, Neuroscience, Limbic system, Epilepsy
References
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- Amnesia produced in cats by local seizure activity initiated from the amygdala.
- Functional alteration of deep structures in cats with chronic focal cortical irritative lesions.
- Primary and secondary spike foci following depth lesions.
- Secondary Epileptogenic Lesions
- Production of epileptiform afterdischarges in cat's cerebral cortex.
- Experimental Epilepsy in the Mouse.∗
- The experimental production of epilepsy in animals.
- Epilépsy; Psychiatric Aspects of Convulsive Disorders
Cited by
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- Pharmacological prophylaxis in the kindling model of epilepsy.
- Mood Disorders in Patients with Epilepsy
- Neonatal seizures associated with narcotic withdrawal.
- Upregulation of NMDA Receptors in Hippocampus and Cortex in the Pentylenetetrazol-Induced “Kindling” Model of Epilepsy
- Recurrent Spontaneous Seizure State Induced by Prefrontal Kindling in Senegalese Baboons, Papio Papio
- Amygdala kindling does not alter the N-methyl-D-aspartate receptor-channel complex which modulates dopamine release in the rat striatum and amygdala.
- NMDA receptor-mediated excitability in dendritically deformed dentate granule cells in pilocarpine-treated rats.
- Evaluation of Amino-Oxyacetic Acid as a Palliative in Tinnitus
- Nifedipine has paradoxical effects on the development of kindling but not on kindled seizures in amygdala-kindled rats.
- Sensitization and Kindling Perspectives for the Course of Affective Illness: Toward a New Treatment with the Anticonvulsant Carbamazepine
- Epilepsy-based changes in hippocampal excitability: causes and effects.
- The NMDA antagonist, MK-801, suppresses long-term potentiation, kindling, and kindling-induced potentiation in the perforant path of the unanesthetized rat.
- Opposite changes of dopamine turnover in prefrontal cortex and nucleus accumbens after amygdaloid kindling.
- From experiences of medial amygdalotomy on epileptics.
- Inositol 1-phosphate formation in long-term potentiation and kindling.
- The effect of hypophysectomy, ACTH fragments and thalamic lesions upon kindled epilepsy.
- Inhibitory processes in development of seizure activity in hippocampal slices
- Psychopathology and personality characteristics of epileptic patients: epilepsy, psychopathology and personality
- Long-term inhibition of kindled seizures by chemical and electrophysiological techniques: insights into the kindling process?
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