Association study between keratinocyte‐derived growth factor gene polymorphisms and susceptibility to vitiligo vulgaris in a Taiwanese population: potential involvement of stem cell factor
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Summary
Vitiligo vulgaris is a depigmentary disorder resulting from the disappearance of functional melanocytes and the pathogenesis of this disorder remains obscure.
- Type
- article
- Published
- 2009-06-01
- Cited by
- 12
- References
- 69
- OpenAlex
- https://openalex.org/W2017542684
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:19077118
Keywords
Vitiligo, Keratinocyte, Biology, Genetics, Gene
References
- Serum stem cell growth factor for monitoring hematopoietic recovery following stem cell transplantation
- FK506 promotes melanocyte and melanoblast growth and creates a favourable milieu for cell migration via keratinocytes: possible mechanisms of how tacrolimus ointment induces repigmentation in patients with vitiligo
- HLA class II haplotype DRB1*04-DQB1*0301 contributes to risk of familial generalized vitiligo and early disease onset.
- PTPN22 is genetically associated with risk of generalized vitiligo, but CTLA4 is not.
- In utero manipulation of coat color formation by a monoclonal anti‐c‐kit antibody: two distinct waves of c‐kit‐dependency during melanocyte development.
- Genetic epidemiology of vitiligo: multilocus recessivity cross-validated.
- Candidate functional promoter variant in the FOXD3 melanoblast developmental regulator gene in autosomal dominant vitiligo.
- A genetical model for vitiligo.
- Contiguous patterns of c-kit and steel expression: analysis of mutations at the W and Sl loci.
- Signalling mechanisms of endothelin-induced mitogenesis and melanogenesis in human melanocytes.
- TRP-2/DT, a new early melanoblast marker, shows that steel growth factor (c-kit ligand) is a survival factor.
- Studies on vitiligo. II. Familial aggregation and genetics
- Epidemiology of vitiligo and associated autoimmune diseases in Caucasian probands and their families.
- Linkage and association of HLA class II genes with vitiligo in a Dutch population
- Evidence for a susceptibility gene, SLEV1, on chromosome 17p13 in families with vitiligo-related systemic lupus erythematosus.
- The biology of melanocytes.
- Cloning, expression, and characterization of a cDNA encoding a novel human growth factor for primitive hematopoietic progenitor cells.
- Association of HLA class I alleles with vitiligo in Chinese Hans.
- NALP1 in vitiligo-associated multiple autoimmune disease.
- Keratinocyte cultures from involved skin in vitiligo patients show an impaired in vitro behaviour.
Cited by
- The association of functional polymorphisms in the aryl hydrocarbon receptor (AHR) gene with the risk of vitiligo in Han Chinese populations
- An Investigation of Autoantibodies against Tyrosine Hydroxylase in Patients with Vitiligo
- Activation of the unfolded protein response in vitiligo: the missing link?
- Comprehensive Association Analysis of Candidate Genes for Generalized Vitiligo Supports XBP1, FOXP3, and TSLP
- Endothelin-1 gene polymorphism in Egyptian patients with vitiligo
- Molecular structure, expression, and functional role of Clec11a in skeletal biology and cancers
- Down‐regulation of exosomal miR‐200c derived from keratinocytes in vitiligo lesions suppresses melanogenesis
- The Modern View on Etiopathogenesis of Vitiligo. Literature Review
- Mitochondria–Melanocyte cellular interactions: An emerging mechanism of vitiligo pathogenesis
- Genetic perspectives on the influence of circulating cytokines on acne: A Mendelian randomization study
- The role of aryl hydrocarbon receptor in vitiligo: a review
- Discovery and Replication of Pathway-Based Trans-Expression Quantitative Trait Loci
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