The development of emphysema in cigarette smoke-exposed mice is strain dependent.
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Summary
Lm, elastance, and inflammation are features that are needed to phenotype emphysema in mice, and the inflammatory cell and cytokine profile may be an important determinant of the phenotype in response to cigarette smoke exposure.
- Type
- article
- Published
- 2004-11-01
- Cited by
- 284
- References
- 42
- OpenAlex
- https://openalex.org/W1999450087
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:8236576
Keywords
Inflammation, Medicine, Phenotype, COPD, Immunology
References
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Cited by
- 3-Week Inhalation Exposure to Cigarette Smoke and/or Lipopolysaccharide in AKR/J Mice
- Mechanisms of cigarette smoke-induced COPD: insights from animal models.
- The role of animal models in the pharmacological evaluation of emerging anti-inflammatory agents for the treatment of COPD.
- Animal models of chronic obstructive pulmonary disease
- Improvement of animal models of COPD and identification of new therapeutic intervention principles therein
- Tomato juice prevents from developing emphysema induced by chronic exposure to tobacco smoke in senescence-accelerated mouse P1 strain.
- Mechanisms of Cigarette Smoke-Induced Inflammation and the Exacerbated Response to Bacteria in Mice
- Effect of inhaled corticosteroids on viral and bacterial infection in chronic obstructive pulmonary disease
- Lung emphysema induced by cigarette smoke: Studies in mice
- Protein carbonylation and muscle function in COPD and other conditions.
- The pathogenesis of lung emphysema: lessons learned from murine models
- Chronic smoke exposure induces rheumatoid factor and anti‐heat shock protein 70 autoantibodies in susceptible mice and humans with lung disease
- IL-18 Is Induced and IL-18 Receptor α Plays a Critical Role in the Pathogenesis of Cigarette Smoke-Induced Pulmonary Emphysema and Inflammation1
- Transcriptional profiling and targeted proteomics reveals common molecular changes associated with cigarette smoke-induced lung emphysema development in five susceptible mouse strains
- Angiotensin-converting enzyme I/D polymorphism in chronic obstructive pulmonary disease
- Cigarette smoke and ozone effect on murine inflammatory responses
- Cigarette Smoke Exposure as a Model of Inflammation Associated with COPD
- Micro-imaging of the Mouse Lung via MRI
- Influence of smoking on airway inflammation and remodelling in asthma
- Modelo experimental de indução de enfisema pulmonar por exposição à fumaça de cigarro.
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