Induction of human interleukin-1 by a product of Staphylococcus aureus associated with toxic shock syndrome.
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Summary
It is concluded that in TSS the sudden fever and probably other components of the acute phase response may be attributed to a massive release of IL-1.
- Type
- article
- Published
- 1984-05-01
- Cited by
- 139
- References
- 21
- Access
- Open access
- OpenAlex
- https://openalex.org/W1986402170
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:12320027
Keywords
Staphylococcus aureus, Toxic shock syndrome, Microbiology, Monocyte, In vitro
References
- Studies in staphylococcal fever. V. Staphlococcal filtrate pyrogen.
- Role of arachidonate metabolism in the immunoregulatory function of human leukocytic pyrogen/lymphocyte-activating factor/interleukin 1.
- Production of staphylococcal enterotoxin F and pyrogenic exotoxin C by Staphylococcus aureus isolates from toxic shock syndrome-associated sources
- Adherent cell function in murine T-lymphocyte antigen recognition. IV. Enhancement of murine T-cell antigen recognition by human leukocytic pyrogen
- Biological effects of bacterial endotoxins in man.
- The role of macrophages in the acute-phase response: SAA inducer is closely related to lymphocyte activating factor and endogenous pyrogen.
- Genetic studies on Staphylococcal strains from patients with toxic shock syndrome.
- Ability of human leukocytic pyrogen to enhance phytohemagglutinin induced murine thymocyte proliferation.
- Toxin and enzyme characterization of Staphylococcus aureus isolates from patients with and without toxic shock syndrome.
- Molecular basis of fever in humans.
- The toxic shock syndrome exotoxin structural gene is not detectably transmitted by a prophage
- POTENTIATION OF THE T-LYMPHOCYTE RESPONSE TO MITOGENS
- A new staphylococcal enterotoxin, enterotoxin F, associated with toxic-shock-syndrome Staphylococcus aureus isolates.
- Staphylococcal pyrogenic exotoxin type C: further characterization.
- Purification and physicochemical and biological characterization of a staphylococcal pyrogenic exotoxin
- QUANTITATIVE ASPECTS OF THE PYROGENIC RESPONSE OF RABBITS TO ENDOTOXIN.
- Further studies on the antipyretic action of polymyxin B in pyrogen-induced fever.
- Molecular basis of fever in humans
Cited by
- Toxic shock syndrome toxin-1, toxic shock, and the immune system.
- Retrovirus-Superantigen-Hypothese der Multiplen Sklerose
- Superantigens and their potential role in human disease.
- Monoclonal antibodies and superantigens: A novel therapeutic approach
- Preparation of protein A immobilized on collodion-coated charcoal and plasma perfusion system for treatment of cancer.
- Chick embryo assay for staphylococcal toxic shock syndrome toxin-1.
- The immunomodulatory function of staphylococcal superantigen on oral tolerance
- Staphylococcus aureus delta toxin as an enterotoxin.
- Study of the Biological Activities of Toxic Shock Syndrome Toxin‐1
- Competitive, enzyme-linked immunosorbent assay for toxic shock syndrome toxin 1
- Staphylococcal Protein A column: correlation of mitogenicity of perfused plasma with clinical response.
- Lymphocyte activation gene-3, a MHC class II ligand expressed on activated T cells, stimulates TNF-alpha and IL-12 production by monocytes and dendritic cells.
- Established macrophagelike cell lines synthesize interleukin-1 in response to toxic shock syndrome toxin
- Immunological protection of rabbits infected with Staphylococcus aureus isolates from patients with toxic shock syndrome
- Role of antigen-presenting cells in activation of human T cells by the streptococcal M protein superantigen: requirement for secreted and membrane-associated costimulatory factors
- Release of interleukin-1 beta associated with potent cytocidal action of staphylococcal alpha-toxin on human monocytes
- Interleukin-1 is linked to the respiratory epithelial cytopathology of pertussis
- Fluid replacement protection of rabbits challenged subcutaneous with toxic shock syndrome toxins
- Toxicity of staphylococcal enterotoxins potentiated by lipopolysaccharide: major histocompatibility complex class II molecule dependency and cytokine release
- A toxic shock syndrome toxin 1 mutant that defines a functional site critical for T-cell activation
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