A protein related to extracellular matrix proteins deleted in the mouse mutant reeler
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Summary
The reeler phenotype seems to reflect a failure of early events associated with brain lamination which are normally controlled by reelin.
- Type
- article
- Published
- 1995-04-20
- Cited by
- 1,759
- References
- 22
- OpenAlex
- https://openalex.org/W1979890518
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:4266946
Keywords
Reeler, Mutant, Extracellular matrix, Extracellular, Biology
References
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- A Method for Isolation of Intact, Translationally Active Ribonucleic Acid
- Chlorambucil-induced mutations in mice recovered in homozygotes.
- Observations on the cerebellum of normal‐reeler mutant mouse chimera
- Analysis of proopiomelanocortin gene expression during prenatal development of the rat pituitary gland.
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- New Neurochemical Markers for Psychosis: A Working Hypothesis of Their Operation
- Inherited hearing defects in mice.
- Neuropathologic findings in a case of OFDS type VI (Váradi syndrome).
- Prenatal effects of drugs of abuse on brain development.
- Expression and chromosomal localization of KIAA0369, a putative kinase structurally related to Doublecortin
- Down-regulated expression of glutamate transporter GLAST in Purkinje cell-associated astrocytes of reeler and weaver mutant cerebella.
- Reelin immunoreactivity in dissociated cultures of the postnatal hippocampus.
- Neuronal Migration, Cerebral Cortical Development, and Cerebral Cortical Anomalies
- Perspectives in the Study of Thyroid Hormone Action on Brain Development and Function
- Cellular and genetic regulation of the development of the cerebellar system.
- Phosphoproteomic Analysis of the Developing Mouse Brain*S
- The human reelin gene: transcription factors (+), repressors (-) and the methylation switch (+/-) in schizophrenia.
- A stream of cells migrating from the caudal telencephalon reveals a link between the amygdala and neocortex
- Analysis of Potential Biomarkers and Modifier Genes Affecting the Clinical Course of CLN3 Disease
- Regulation of neural migration by the CREB/CREM transcription factors and altered Dab1 levels in CREB/CREM mutants.
- Reelin haploinsufficiency reduces the density of PV+ neurons in circumscribed regions of the striatum and selectively alters striatal-based behaviors
- Lack of reelin modifies the gene expression in the small intestine of mice
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