Regulation of Ca2+-activated nonselective cationic currents in rat pituitary GH3 cells: involvement in L-type Ca2+ current.
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Summary
The present studies clearly indicate that Ca2+-activated nonselective cationic channels are expressed in GH3 cells, and can be elicited by the depolarizing stimuli that lead to the activation of ICa,L.
- Type
- article
- Published
- 1998-11-23
- Cited by
- 29
- References
- 17
- OpenAlex
- https://openalex.org/W1978038878
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:19348225
Keywords
Depolarization, Repolarization, Chemistry, Biophysics, EGTA
References
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- Uncoupling of Calcium Mobilization and Entry Pathways in Endothelin-stimulated Pituitary Lactotrophs*
- Ion channels in human neutrophils activated by a rise in free cytosolic calcium concentration
- Single Ca2+-activated nonselective cation channels in neuroblastoma
- Effect of capsaicin on membrane currents in cultured vascular smooth muscle cells of rat aorta.
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- Non-selective cation channel on pancreatic duct cells.
- Ca(2+)-dependent and Ca(2+)-independent mechanisms modulate whole-cell cationic currents in human neutrophils.
- Calcium-activated non-specific cation channels.
- Ionic mechanisms of tetrandrine in cultured rat aortic smooth muscle cells.
- Action potentials and membrane ion channels in clonal anterior pituitary cells.
- Mechanism of thyrotropin releasing hormone stimulation of pituitary hormone secretion.
- A Long-Lasting Calcium-Activated Nonselective Cationic Current Is Generated by Synaptic Stimulation or Exogenous Activation of Group I Metabotropic Glutamate Receptors in CA1 Pyramidal Neurons
- Alterations in the frequency and shape of Ca2+ fluctuations in GH4C1 cells induced by thyrotropin-releasing hormone and Bay K 8644.
- Calcium-dependent plateau potentials in a crab stomatogastric ganglion motor neuron. II. Calcium-activated slow inward current.
- Pharmacological characterization of two calcium currents in GH3 cells.
Cited by
- Inhibition of endoplasmic reticulum Ca²⁺ ATPase in preBötzinger complex of neonatal rat does not affect respiratory rhythm generation.
- Dependence of the Excitability of Pituitary Cells on Cyclic Nucleotides
- On the mechanism of selective action of probucol on the inwardly rectifying potassium current in GH3 lactotrophs
- Vinpocetine-induced stimulation of calcium-activated potassium currents in rat pituitary GH3 cells.
- Ion channels and signaling in the pituitary gland.
- Channel modulators affect PGE(2) binding to bovine aortic endothelial cells.
- Fangchinoline inhibits rat aortic vascular smooth muscle cell proliferation and cell cycle progression through inhibition of ERK1/2 activation and c-fos expression.
- Differential effects of quercetin, a natural polyphenolic flavonoid, on L‐Type calcium current in pituitary tumor (GH3) cells and neuronal NG108‐15 cells
- Ceramide Inhibits the Inwardly Rectifying Potassium Current in GH3 Lactotrophs.
- Role of K+ Channels in Frequency Regulation of Spontaneous Action Potentials in Rat Pituitary GH3 Cells
- The mechanism of actions of 3-(5'-(hydroxymethyl-2'-furyl)-1-benzyl indazole (YC-1) on Ca(2+)-activated K(+) currents in GH(3) lactotrophs.
- Actions of epoxyeicosatrienoic acid on large-conductance Ca(2+)-activated K(+) channels in pituitary GH(3) cells.
- Block of erg current by linoleoylamide, a sleep-inducing agent, in pituitary GH3 cells.
- Tramadol-induced block of hyperpolarization-activated cation current in rat pituitary lactotrophs
- Pituitary cell lines and their endocrine applications.
- Effects of tetrandrine on calcium transport, protein fluorescences and membrane fluidity of sarcoplasmic reticulum
- Characterizing the effects of Eugenol on neuronal ionic currents and hyperexcitability
- Enhanced activity of Ca2+‐activated K+ channels by 1‐[2‐hydroxy‐3‐propyl‐4‐[(1H‐tetrazol‐5‐yl)butoxyl]phenyl] ethanone (LY‐171883) in neuroendocrine and neuroblastoma cell lines
- Inhibitory effect of memantine, an NMDA-receptor antagonist, on electroporation-induced inward currents in pituitary GH3 cells.
- Cav1.2 and Cav1.3 L-type calcium channels operate in a similar voltage range but show different coupling to Ca(2+)-dependent conductances in hippocampal neurons.
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