Defective autophagy in spastizin mutated patients with hereditary spastic paraparesis type 15.

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Summary

Autophagy is a central process in the pathogenesis of complicated forms of hereditary spastic paraparesis with thin corpus callosum and results indicate that autophagy defects with autophagosome accumulation can be observed also in neuronal cells upon spastizin silencing.

Type
article
Published
2013-10-01
Cited by
88
References
75
Access
Open access

Keywords

Autophagy, Cell biology, Biology, Cytokinesis, Endoplasmic reticulum

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