Glutathione S-transferase M1, T1, and P1 genotypes and rheumatoid arthritis.
Explore this paper's citation graph
Summary
The results suggest that the deletion polymorphism of GSTM1 is associated with increased susceptibility for RA, particularly among individuals who are not carriers of the HLA-DRB 1 SE.
- Type
- article
- Published
- 2005-06-01
- Cited by
- 36
- References
- 37
- OpenAlex
- https://openalex.org/W1786488539
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:20443717
Keywords
Medicine, Genotype, GSTP1, Glutathione S-transferase, Odds ratio
References
- Release of oxygen radicals by articular chondrocytes: A study of luminol‐dependent chemiluminescence and hydrogen peroxide secretion
- Psychometric evaluation of a Korean Health Assessment Questionnaire for clinical research.
- PPARgamma ligands as modulators of inflammatory and catabolic responses in arthritis. An overview.
- Glutathione S-transferase polymorphisms and colorectal cancer: a HuGE review.
- Inadequate Antioxidant Nutrient Intake and Altered Plasma Antioxidant Status of Rheumatoid Arthritis Patients
- Susceptibility to ulcerative colitis and Crohn's disease: interactions between glutathione S-transferase GSTM1 and GSTT1 genotypes.
- Correlation between disease phenotype and genetic heterogeneity in rheumatoid arthritis.
- The Influence of HLA-DRB1 Genes on Disease Severity in Rheumatoid Arthritis
- The shared epitope hypothesis. An approach to understanding the molecular genetics of susceptibility to rheumatoid arthritis.
- Homozygosity for the HLA-DRB1 allele selects for extraarticular manifestations in rheumatoid arthritis.
- Antioxidant status in rheumatoid arthritis and role of antioxidant therapy.
- Glutathione S-transferase M1, T1, and P1 gene polymorphisms and carotid atherosclerosis in Korean patients with rheumatoid arthritis
- HLA-DR alleles with naturally occurring amino acid substitutions and risk for development of rheumatoid arthritis.
- The glutathione S-transferase supergene family: regulation of GST and the contribution of the isoenzymes to cancer chemoprotection and drug resistance.
- Oxidant/antioxidant status of plasma samples from patients with rheumatoid arthritis
- Sequencing of HLA class II genes based on the conserved diversity of the non-coding regions: sequencing based typing of HLA-DRB genes.
- Smoking and disease severity in rheumatoid arthritis: association with polymorphism at the glutathione S-transferase M1 locus.
- Ethnic distribution of the glutathione transferase Mu 1-1 (GSTM1) null genotype in 1473 individuals and application to bladder cancer susceptibility.
- Drug derived radicals: mediators of the side effects of anti-inflammatory drugs?
- The genetics revolution and the assault on rheumatoid arthritis.
Cited by
- Genetic polymorphism of glutathione S-transferase T1 and the risk of rheumatoid arthritis: a meta-analysis.
- Association of GSTM1, GSTT1, GSTP1-ILE105VAL and ACE I/D polymorphisms with ankylosing spondylitis
- Transcriptomic analysis of peripheral blood monocytes and synovial macrophages in inflammatory arthritis
- Autoantibodies to glutathione S-transferase theta 1 in patients with primary sclerosing cholangitis and other autoimmune diseases.
- Non-Jewish Israeli IBD Patients Have Significantly Higher Glutathione S-Transferase GSTT1-Null Frequency
- Genetic Polymorphism in Glutathione Transferases (GST): Population Distribution of GSTM1, T1, and P1 Conjugating Activity
- Glutathione S-transferase M1, T1, and P1 polymorphisms as susceptibility factors for noise-induced temporary threshold shift.
- Association analysis of TNFR2, VDR, A2M, GSTT1, GSTM1, and ACE genes with rheumatoid arthritis in South Asians and Caucasians of East Midlands in the United Kingdom
- CYP1A2 genotype and rheumatoid arthritis in Koreans
- Expression patterns and novel splicing variants of glutathione-S-transferase isoenzymes of human lung and hepatocyte cell lines
- Effect of Glutathione S-Transferase T1, M1, P1 and Heme Oxygenase-1 Polymorphisms Interactions with Heavy Smoking on the Risk of Rheumatoid Arthritis
- A Possible Relationship Between Polymorphisms of Glutathione S-Transferase M1, P1 and T1 Genes and Rheumatoid Arthritis in Zahedan, Southeast Iran
- Epigenome-wide association data implicate DNA methylation as an intermediary of genetic risk in Rheumatoid Arthritis
- Associations analysis of GSTM1, T1 and P1 Ile105Val polymorphisms with carpal tunnel syndrome
- Effects of GSTM1 in Rheumatoid Arthritis; Results from the Swedish EIRA study
- Association between the polymorphisms of glutathione S-transferase genes and rheumatoid arthritis: a meta-analysis.
- The Search for Reliable Biomarkers of Disease in Multiple Chemical Sensitivity and Other Environmental Intolerances
- The glutathione S-transferase M1 and P1 polymorphisms and rheumatoid arthritis: a meta-analysis
- Association of glutathione S-transferase gene polymorphisms (GSTM1 and GSTT1) of vitiligo in Korean population.
- Association of Glutatione S-Transferase (GSTM1 and GSTT1) Gene Deletions in Korean Patients with Alcoholism
Related papers
- Association of polymorphism in glutathione S-transferase loci with susceptibility and outcome in rheumatoid arthritis: comparison with the shared epitope
- Genetic polymorphisms of glutathione S-transferases and disease activity of rheumatoid arthritis.
- Smoking and disease severity in rheumatoid arthritis: association with polymorphism at the glutathione S-transferase M1 locus.
- Effect of Glutathione S-Transferase T1, M1, P1 and Heme Oxygenase-1 Polymorphisms Interactions with Heavy Smoking on the Risk of Rheumatoid Arthritis
- Glutathione S-transferase gene polymorphisms in Japanese patients with rheumatoid arthritis.
- Cyclooxygenase-2 genotype and rheumatoid arthritis.
- HLA-DRB1 genotype influences risk for and severity of rheumatoid arthritis.
- Increased susceptibility to rheumatoid arthritis in Koreans heterozygous for HLA-DRB1*0405 and *0901.
- HLA‐DRB1 genes and disease severity in rheumatoid arthritis in Turkey
- The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis.