Vascular-specific growth factors and blood vessel formation
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Summary
New findings in newly discovered vascular growth factors demand re-evaluation of therapeutic efforts aimed at regulating blood vessel growth in ischaemia, cancer and other pathological settings.
- Type
- review
- Published
- 2000-09-14
- Cited by
- 3,872
- References
- 74
- OpenAlex
- https://openalex.org/W1783580437
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:4420263
Keywords
Pathological, Harmony (color), Blood vessel, Medicine, Biology
References
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- Enhanced expression of the tie receptor tyrosine kinase in endothelial cells during neovascularization.
- Analysis of blood vessel maturation processes during cyclic ovarian angiogenesis.
- The chick embryo chorioallantoic membrane as a model for in vivo research on angiogenesis.
- Vascular endothelial growth factor receptor-3.
- Roles of vascular endothelial growth factor and astrocyte degeneration in the genesis of retinopathy of prematurity.
- Structure, expression and receptor-binding properties of placenta growth factor (PlGF).
- Vascular permeability factor/vascular endothelial growth factor and the significance of microvascular hyperpermeability in angiogenesis.
- Quantitative immunohistological analysis of the microvasculature in untreated human glioblastoma multiforme. Computer-assisted image analysis of whole-tumor sections.
- Mechanisms of angiogenesis and arteriogenesis
- Non-small-cell lung carcinoma tumor growth without morphological evidence of neo-angiogenesis.
- VEGF couples hypertrophic cartilage remodeling, ossification and angiogenesis during endochondral bone formation
- Vascular endothelial growth factor: molecular and biological aspects.
- A model of angiogenesis in the mouse cornea.
- Angiopoietin-1 protects the adult vasculature against plasma leakage
- Mechanisms of angiogenesis
- Development of retinal vasculature is mediated by hypoxia-induced vascular endothelial growth factor (VEGF) expression by neuroglia
- Clinical applications of angiogenic growth factors and their inhibitors
- Tie-1 and tie-2 define another class of putative receptor tyrosine kinase genes expressed in early embryonic vascular system.
- Inhibition of vascular endothelial growth factor-induced angiogenesis suppresses tumour growth in vivo
Cited by
- Genetic modulation of hypoxia induced gene expression and angiogenesis: relevance to brain tumors.
- Artificial engineering of secondary lymphoid organs.
- Bioimmunotherapeutic targets on angiogenetic blood vessels in solid malignangies.
- Collateral Growth and Angiogenesis Around Cortical Stroke
- Polymeric system for dual growth factor delivery
- Angiopoietin-1, angiopoietin-2 and Tie-2 in tumour and non-tumour tissues during growth of experimental melanoma
- Vitreous fluid biomarkers.
- Lack of collagen XVIII/endostatin results in eye abnormalities
- Vascular endothelial growth factor causally contributes to the angiogenic response upon ultraviolet B irradiation in vivo
- The emerging role of angiogenesis inhibitors in hematologic malignancies.
- Human placental vascular development: vasculogenic and angiogenic (branching and nonbranching) transformation is regulated by vascular endothelial growth factor-A, angiopoietin-1, and angiopoietin-2.
- Suppression of tumor angiogenesis through the inhibition of integrin function and signaling in endothelial cells: which side to target?
- CYR61 (CCN1) Is Essential for Placental Development and Vascular Integrity
- Natural product derived receptor tyrosine kinase inhibitors: identification of IGF1R, Tie-2, and VEGFR-3 inhibitors.
- Inhibition of constitutive NF-κB activity by IκBαM suppresses tumorigenesis
- Angiopoietin‐1 inhibits endothelial permeability, neutrophil adherence and IL‐8 production
- Mechanism of IL-12 mediated alterations in tumour blood vessel morphology: analysis using whole-tissue mounts
- Therapeutic stem and progenitor cell transplantation for organ vascularization and regeneration
- Localization of Tie2 and phospholipase D in endothelial caveolae is involved in angiopoietin-1-induced MEK/ERK phosphorylation and migration in endothelial cells.
- Die Blockade des Neuropilin-2 als mögliches therapeutisches Prinzip bei Arthritis
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