ST2 and IL-33 differentially regulate the innate immune response to Streptococcus pneumoniae
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- Published
- 2015-01-01
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- 0
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- 243
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:83963898
References
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- An IL-1 Cytokine Member, IL-33, Induces Human Basophil Activation via Its ST2 Receptor1
- Innate lymphoid cells — how did we miss them?
- IL‐33 attenuates EAE by suppressing IL‐17 and IFN‐γ production and inducing alternatively activated macrophages
- Alveolar macrophages in pulmonary host defence – the unrecognized role of apoptosis as a mechanism of intracellular bacterial killing
- The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5
- Cutting Edge: Critical Role for Mesothelial Cells in Necrosis-Induced Inflammation through the Recognition of IL-1α Released from Dying Cells1
- IL-33 citrine reporter mice reveal the temporal and spatial expression of IL-33 during allergic lung inflammation
- The Macrophage-Inducible C-Type Lectin, Mincle, Is an Essential Component of the Innate Immune Response to Candida albicans1
- Recognition of Streptococcus pneumoniae by the innate immune system
- Cutting Edge: Mincle Is Essential for Recognition and Adjuvanticity of the Mycobacterial Cord Factor and its Synthetic Analog Trehalose-Dibehenate
- Alveolar Macrophage Apoptosis Contributes to Pneumococcal Clearance in a Resolving Model of Pulmonary Infection 1
- Reduced virulence of a defined pneumolysin-negative mutant of Streptococcus pneumoniae
- Expression and regulation of interleukin‐33 in human monocytes
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