Evidence for Phagocytosis of Influenza Virus-Infected, Apoptotic Cells by Neutrophils and Macrophages in Mice1
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Summary
The results suggested that the level of phagocytosis is augmented both quantitatively and qualitatively in the lung of influenza virus-infected animals so that infected cells are effectively eliminated.
- Type
- article
- Published
- 2007-02-15
- Cited by
- 246
- References
- 42
- Access
- Open access
- OpenAlex
- https://openalex.org/W1575690521
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:26011964
Keywords
Phagocytosis, Apoptosis, Microbiology, Virus, Biology
References
- Role of Phosphatidylserine Exposure and Sugar Chain Desialylation at the Surface of Influenza Virus-infected Cells in Efficient Phagocytosis by Macrophages*
- Phagocytosis: elegant complexity.
- Surfactant Protein A Enhances Alveolar Macrophage Phagocytosis of Apoptotic Neutrophils1
- Apoptosis: a mechanism of cell killing by influenza A and B viruses
- The curious world of apoptotic cell clearance
- Inhibitory Effect of Toll-Like Receptor 4 on Fusion between Phagosomes and Endosomes/Lysosomes in Macrophages1
- Pathogen recognition and innate immunity.
- Promotion of the uptake of PS liposomes and apoptotic cells by a product of growth arrest-specific gene, gas6.
- Identification of a factor that links apoptotic cells to phagocytes
- Toll-like receptor 4 is not involved in host defense against respiratory tract infection with Sendai virus
- Phosphatidylserine Binding of Class B Scavenger Receptor Type I, a Phagocytosis Receptor of Testicular Sertoli Cells*
- Identification of Proteins Cleaved Downstream of Caspase Activation in Monocytes Undergoing Macrophage Differentiation*
- Augmentation of fatality of influenza in mice by inhibition of phagocytosis.
- Co-expression of Fas and Fas-ligand on the surface of influenza virus-infected cells
- Ringing the alarm bells: signalling and apoptosis in influenza virus infected cells
- ESTERASES IN HUMAN LEUKOCYTES
- The interface between innate and adaptive immunity
- Programmed cell death (apoptosis) in human monocytes infected by influenza A virus.
- Lectin-like oxidized low-density lipoprotein receptor 1 mediates phagocytosis of aged/apoptotic cells in endothelial cells.
- Phosphatidylserine-Mediated Phagocytosis of Influenza A Virus-Infected Cells by Mouse Peritoneal Macrophages
Cited by
- Immune Responses to Influenza Virus and Its Correlation to Age and Inherited Factors
- Induction of Apoptosis and Subsequent Phagocytosis of Virus-Infected Cells As an Antiviral Mechanism
- Innate Immune Sensing and Response to Influenza
- The role of pulmonary dendritic cells in regulating the antigen-specific CD8 T cell response following influenza virus infection
- IL-17 Modulation of Rhinovirus-Induced Allergic Airways Disease
- Influenza virus infection in a compromised immune system
- Gimme shelter: the immune system during pregnancy
- Current Status of Monocyte Differentiation-Inducing (MDI) Factors Derived from Human Fetal Membrane Chorion Cells Undergoing Apoptosis after Influenza Virus Infection
- The Mouse as Model System to Study Host‐Pathogen Interactions in Influenza A Infections
- INFLUENZA SPECIFIC T- AND B-CELL RESPONSES IN IMMUNOSUPPRESSED PATIENTS
- A comparison of both water and ethanol extracts prepared from Echinacea purpurea and Echinacea angustifolia on the response to Influenza A/PR/8/34 infection in mice
- Transient ablation of alveolar macrophages leads to massive pathology of influenza infection without affecting cellular adaptive immunity
- Universal Vaccine Based on Ectodomain of Matrix Protein 2 of Influenza A: Fc Receptors and Alveolar Macrophages Mediate Protection
- Phagocytic removal of cells that have become unwanted: Implications for animal development and tissue homeostasis
- Mapping the pulmonary environment of animals protected from virulent H1N1 influenza infection using the TLR‐2 agonist Pam2Cys
- Efferocytosis is an Innate Antibacterial Mechanism of Mycobacterium tuberculosis Control
- Mechanisms of Cross-protection by Influenza Virus M2-based Vaccines
- Interactions of α-, β-, and θ-Defensins with Influenza A Virus and Surfactant Protein D
- 17β-Estradiol Protects Females against Influenza by Recruiting Neutrophils and Increasing Virus-Specific CD8 T Cell Responses in the Lungs
- Responses of Mouse Airway Epithelial Cells and Alveolar Macrophages to Virulent and Avirulent Strains of Influenza A Virus
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