Mechanisms responsible for homocysteine mediated damage to human endothelial cells : the role of oxidative stress in atherogenesis
Explore this paper's citation graph
Summary
The next chapter discusses conference attendance and presentation techniques, as well as some of the concepts used in the presentation.
- Type
- dissertation
- Published
- 2009-01-01
- Cited by
- 0
- References
- 434
- OpenAlex
- https://openalex.org/W858084474
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:82029037
Keywords
Oxidative stress, Homocysteine, Endothelial dysfunction, Endothelium, Oxidative damage
References
- Detection of superoxide generated by endothelial cells.
- Neutrophils roll on adherent neutrophils bound to cytokine-induced endothelial cells via L-selectin on the rolling cells
- Tumor Necrosis Factor α-Induced E-selectin Expression Is Activated by the Nuclear Factor-κB and c-JUN N-terminal Kinase/p38 Mitogen-activated Protein Kinase Pathways*
- Homocyst(e)ine Decreases Bioavailable Nitric Oxide by a Mechanism Involving Glutathione Peroxidase*
- Characterization of the stress-inducing effects of homocysteine.
- Activation of JNK and transcriptional repressor ATF3/LRF1 through the IRE1/TRAF2 pathway is implicated in human vascular endothelial cell death by homocysteine.
- Homocysteine activates NADH/NADPH oxidase through ceramide-stimulated Rac GTPase activity in rat mesangial cells.
- Neuronal nitric oxide synthase (nNOS) modulates the JNK1 activity through redox mechanism: a cGMP independent pathway.
- Homocysteine induces endothelial dysfunction via inhibition of arginine transport.
- Inhibition of NF-kappa B by S-nitrosylation.
- Immunohistochemical Study of the Phosphorylated and Activated Form of c-Jun NH2-terminal Kinase in Human Aorta
- The biochemical basis of nitroblue tetrazolium reduction in normal human and chronic granulomatous disease polymorphonuclear leukocytes.
- Vascular pathology of homocysteinemia: implications for the pathogenesis of arteriosclerosis.
- Homocysteine and thrombotic disease.
- Leukocyte-endothelial adhesion molecules.
- Intact and altered endothelium in regulation of vasomotion.
- N-acetylcysteine in acute cardiology: 10 years later: what do we know and what would we like to know?!
- Interleukin 8 as a novel target for intervention therapy in acute inflammatory diseases.
- Endotoxin and tumor necrosis factor induce interleukin-1 gene expression in adult human vascular endothelial cells.
- Tetrahydrobiopterin: regulator of endothelial nitric oxide synthase in vascular disease.
Cited by
No citing papers recorded for this paper.
Related papers
- Role of MicroRNAs in homocysteine induced oxidative stress
- Homocysteine and Oxidative Mechanisms of Vascular Remodeling
- THE INTERACTION BETWEEN OXIDATIVE STRESS AND BIOMARKERS OF INFLAMMATION IN ATHEROSCLEROSIS
- The role of nitric oxide and oxidative stress as modulating factors of inflammation in atherosclerosis
- Roles of Vascular Oxidative Stress and Nitric Oxide in the Pathogenesis of Atherosclerosis
- Oxidative stress in the pathogenesis/treatment of atherosclerosis