Homologous interference mediated by defective interfering influenza virus derived from a temperature-sensitive mutant of influenza virus
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Summary
The data suggest that the loss of infectivity observed among DI viruses may be due to nonspecific loss of a viral RNA segment(s), and the interfering property of DI viruses is due to interfering RNA segments (DIRNA, D1 to D6).
- Type
- article
- Published
- 1978-10-01
- Cited by
- 86
- References
- 29
- Access
- Open access
- OpenAlex
- https://openalex.org/W2112637120
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:23208838
Keywords
Virus, Biology, Infectivity, Virology, Viral Interference
References
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- Ribonucleic acid synthesis of vesicular stomatitis virus. VI. Correlation of defective particle RNA synthesis with standard RNA replication.
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- Defective Particles in BHK Cells Infected with Temperature-Sensitive Mutants of Vesicular Stomatitis Virus
- Ribonucleic Acid Synthesis of Vesicular Stomatitis Virus
- Transcriptase activity and genome composition of defective influenza virus
- Viral pathogenesis and molecular biology.
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- Polypeptide composition of incomplete influenza virus grown in MDBK cells.
- Properties of incomplete Sendai virions and subgenomic viral RNAs.
- Sequence relationships between the genome and the intracellular RNA species of standard and defective-interfering Semliki Forest virus.
- Isolation of an influenza virus temperature-sensitive mutant of a new recombination-complementation group.
- The RNA of defective vesicular stomatitis virus particles in relation to viral cistrons.
- The RNAs of infective and incomplete influenza virions grown in MDBK and HeLa cells.
Cited by
- Origin of small RNA in von Magnus particles of influenza virus
- Biochemistry of arenaviruses.
- Origin and replication of defective interfering particles.
- Defective-interfering (DI) RNAs of influenza viruses: origin, structure, expression, and interference.
- Mécanisme d’incorporation des segments d’ARNv d’un virus influenza A aviaire : comparaison avec un virus humain et implication pour le réassortiment génétique
- Defective Influenza Viral Ribonucleoproteins Cause Interference
- Extra RNAs of von Magnus particles of influenza virus cause reduction of particular polymerase genes
- In vitro transcription of defective interfering particles of influenza virus produces polyadenylic acid-containing complementary RNAs
- Incomplete influenza virus: partial functional complementation as revealed by hemadsorbing cell count test
- Complete nucleotide sequence of the polymerase 3 gene of human influenza virus A/WSN/33
- Transmembrane domain of influenza virus neuraminidase, a type II protein, possesses an apical sorting signal in polarized MDCK cells
- Intracellular localization of the viral polymerase proteins in cells infected with influenza virus and cells expressing PB1 protein from cloned cDNA
- Function of two discrete regions is required for nuclear localization of polymerase basic protein 1 of A/WSN/33 influenza virus (H1 N1)
- Formation of influenza virus particles lacking hemagglutinin on the viral envelope
- Influence of the host cell on the genomic and subgenomic RNA content of defective-interfering influenza virus.
- Novel polypeptides encoded by influenza virus subgenomic (DI type) virion RNAs.
- Construction and characterization of a bacterial clone containing the hemagglutinin gene of the WSN strain (HON1) of influenza virus.
- Influenza Virus Subpopulations: Exchange of Lethal H5N1 Virus NS for H1N1 Virus NS Triggers De Novo Generation of Defective-Interfering Particles and Enhances Interferon-Inducing Particle Efficiency
- Temperature-sensitive influenza A virus clones originated by a cross between A/Aichi/2/68 (H3N2) and B/Yamagata/1/73
- Temperature-sensitive mutants of influenza A/Udorn/72 (H3N2) virus. II. Genetic analysis and demonstration of intrasegmental complementation.
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