ANG II potentiates mitogenic effect of norepinephrine in vascular muscle cells: role of FGF-2.
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Summary
It is demonstrated that ANG II and NE cooperate in promoting VSMC growth and that FGF-2 upregulation is involved in this effect.
- Type
- article
- Published
- 2001-01-01
- Cited by
- 13
- References
- 36
- OpenAlex
- https://openalex.org/W2180840815
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:21330635
Keywords
Vascular smooth muscle, Angiotensin II, Internal medicine, Endocrinology, Losartan
References
- Angiotensin II induces transcription and expression of alpha 1-adrenergic receptors in vascular smooth muscle cells.
- Basic fibroblast growth factor overexpression in endothelial cells: an autocrine mechanism for angiogenesis and angioproliferative diseases.
- Repetitive coronary artery occlusions induce release of growth factors into the myocardial interstitium.
- Alpha 1-adrenergic stimulation of platelet-derived growth factor A-chain gene expression in rat aorta.
- TGF-beta s stimulate cell proliferation via an autocrine production of FGF-2 in corneal stromal fibroblasts.
- Effects of an Angiotensin II Receptor Antagonist, CV‐11974, on Angiotensin II‐Induced Increases in Cytosolic Free Calcium Concentration, Hyperplasia, and Hypertrophy of Cultured Vascular Smooth Muscle Cells
- Estrogen stimulates delayed mitogen-activated protein kinase activity in human endothelial cells via an autocrine loop that involves basic fibroblast growth factor.
- Induction of platelet-derived growth factor A-chain and c-myc gene expressions by angiotensin II in cultured rat vascular smooth muscle cells.
- Clenbuterol induces growth factor mRNA, activates astrocytes, and protects rat brain tissue against ischemic damage.
- Angiotensin II stimulates the pp44 and pp42 mitogen-activated protein kinases in cultured rat aortic smooth muscle cells.
- Angiotensin II-stimulated protein synthesis in cultured vascular smooth muscle cells.
- The p42/p44 Mitogen-activated Protein Kinase Cascade Is Determinant in Mediating Activation of the Na+/H+ Exchanger (NHE1 Isoform) in Response to Growth Factors*
- Peptide vasoconstrictors, vessel structure, and vascular smooth-muscle proliferation.
- Angiotensin II responses of vascular smooth muscle cells from hypertensive rats: enhancement at the level of p42 and p44 mitogen activated protein kinase
- Multiple autocrine growth factors modulate vascular smooth muscle cell growth response to angiotensin II.
- Angiotensin II induces smooth muscle cell proliferation in the normal and injured rat arterial wall.
- Angiotensin II and endothelin-1 increase fibroblast growth factor-2 mRNA expression in vascular smooth muscle cells.
- Angiotensin II: Vasoconstrictor or Growth Factor?
- Mitogen-activated protein kinase pathways.
- Mechanism of catecholamine-induced proliferation of vascular smooth muscle cells.
Cited by
- Angiotensin II type 2 receptor gene deficiency attenuates susceptibility to tobacco-specific nitrosamine-induced lung tumorigenesis: involvement of transforming growth factor-beta-dependent cell growth attenuation.
- The atherogenic potential of dietary carbohydrate.
- Heparin recovers AT1 receptor and its intracellular signal transduction in cultured vascular smooth muscle cells
- Angiotensin II induces proliferation of human cerebral artery smooth muscle cells through a basic fibroblast growth factor (bFGF) dependent mechanism.
- Trophic effects induced by alpha1D-adrenoceptors on endothelial cells are potentiated by hypoxia.
- Angiotensin-(1-7) Changes Apoptosis-Related Genes Expression in Human Breast Cancer Cell Line T47D
- Pathogenesis and etiology of essential hypertension: role of dietary carbohydrate.
- Endogenous VEGF-A is responsible for mitogenic effects of MCP-1 on vascular smooth muscle cells.
- Improvement of Vascular Remodeling in Spontaneous Hypertensive Rats with Traditional Chinese Medicine
- Inhibition of the proliferation of smooth muscle cells from human coronary bypass vessels by vasonatrin peptide.
- Natriuretic peptide receptor-C activation regulates vascular smooth muscle cell hypertrophy : molecular mechanisms
- Bibliography Abdelrahim, M. and Safe, S. 2005. Cyclooxygenase-2 Inhibitors Decrease Vascular Endothelial Growth Factor Expression in Colon Cancer Cells by Enhanced Degradation of Sp 1 and Sp 4
- Angiotensin-( 1-7 ) Changes Apoptosis-Related Genes Expression in Human Breast Cancer Cell Line T 47 D
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