Induction of Apoptosis in Rat Myocardium under Anoxic Conditions
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Summary
All changes described here reflect two opposing processes occurring in mitochondria: apoptotic destruction and compensatory processes responsible for maintenance of mitochondria.
- Type
- article
- Published
- 2002-02-01
- Cited by
- 10
- References
- 16
- OpenAlex
- https://openalex.org/W120216451
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:25246472
Keywords
Mitochondrion, Fragmentation (computing), Biology, Population, Anoxic waters
References
- Programmed cell death.
- Phenoptosis: programmed death of an organism.
- Conformational basis of energy transformations in membrane systems. 3. Configurational changes in the mitochondrial inner membrane induced by changes in functional states.
- Tumor Necrosis Factor-α Increases the Steady-state Reduction of Cytochrome b of the Mitochondrial Respiratory Chain in Metabolically Inhibited L929 Cells*
- Preservation of Mitochondrial Structure and Function after Bid- or Bax-Mediated Cytochrome c Release
- Unresolved issues regarding the role of apoptosis in the pathogenesis of ischemic injury and heart failure.
- Apoptosis: A Basic Biological Phenomenon with Wide-ranging Implications in Tissue Kinetics
- Increased apoptosis in the heart of genetic hypertension, associated with increased fibroblasts.
- Hypoxia death stimulus induces translocation of p53 protein to mitochondria
- The Pro-Apoptotic Proteins, Bid and Bax, Cause a Limited Permeabilization of the Mitochondrial Outer Membrane That Is Enhanced by Cytosol
- Normal and abnormal consequences of apoptosis in the human heart: from postnatal morphogenesis to paroxysmal arrhythmias.
- "Apoptotic" myocytes in infarct area in rabbit hearts may be oncotic myocytes with DNA fragmentation: analysis by immunogold electron microscopy combined with In situ nick end-labeling.
- Mitochondrial Events in the Life and Death of Animal Cells: A Brief Overview
- Apoptosis and pleomorphic micromitochondriosis in the sinus nodes surgically excised from five patients with the long QT syndrome.
- Tumor necrosis factor-a induces apoptosis via inducible nitric oxide synthase in neonatal mouse cardiomyocytes
Cited by
- Effect of the inhibitory neurotransmitter glycine on slow destructive processes in brain cortex slices under anoxic conditions
- Functional Activity and Ultrastructure of Mitochondria Isolated from Myocardial Apoptotic Tissue
- Ultrastructural changes in blood cells in the hematopoietic organs of Lake Baikal fish exposed to phenol
- Fusion, Fragmentation, and Fission of Mitochondria
- Study of three-dimensionally ordered structures of intact mitochondria by small-angle neutron scattering
- Electron microscopy description of cardiomyocytes from the left ventricle of rat heart after apoptosis induction by isoproterenol
- Structural organization of nanomaterials and nanosystems: neutron scattering insight
- Thread-grain transition of mitochondrial reticulum as a step of mitoptosis and apoptosis
- Structural and dynamic changes in mitochondria of rat myocardium under acute hypoxic hypoxia: Role of mitochondrial ATP-dependent potassium channel
- Significance of metabolomic disorders in infertility and miscarriage in patients with endometriosis
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