DNA Sequence Variants in Human Autoimmune Diseases
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Summary
By participating in transcription complex with other co-factors, IRF5 and STAT4 harbour the potential of regulating a large number of target genes, which may contribute to their strong association with SLE.
- Published
- 2012-01-01
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- 0
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- 228
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:80955945
References
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- Genetic variants and disease-associated factors contribute to enhanced IRF-5 expression in blood cells of systemic lupus erythematosus patients
- Differential Activation of IFN Regulatory Factor (IRF)-3 and IRF-5 Transcription Factors during Viral Infection1
- Variant form of STAT4 is associated with primary Sjögren's syndrome
- ChIP-seq accurately predicts tissue-specific activity of enhancers
- Interferon regulatory factor 5, a novel mediator of cell cycle arrest and cell death.
- Protection of Irf5-deficient mice from pristane-induced lupus involves altered cytokine production and class switching
- Differential utilization of Janus kinase-signal transducer activator of transcription signaling pathways in the stimulation of human natural killer cells by IL-2, IL-12, and IFN-alpha.
- INSIGHTS FROM GENOMIC PROFILING OF TRANSCRIPTION FACTORS
- Detecting shared pathogenesis from the shared genetics of immune-related diseases
- Multiple polymorphisms in the TNFAIP3 region are independently associated with systemic lupus erythematosus
- IFN-regulatory factor 3-dependent gene expression is defective in Tbk1-deficient mouse embryonic fibroblasts
- Genome-wide association study identifies 12 new susceptibility loci for primary biliary cirrhosis
- IRF5 promotes inflammatory macrophage polarization and TH1-TH17 responses
- Structural Basis for Recognition of the Intron Branch Site RNA by Splicing Factor 1
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