5 Drugs that Target Sympathetic- Immune Pathways for Treatment of Autoimmune Diseases
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- Type
- article
- Published
- 2008-01-01
- Cited by
- 0
- References
- 244
- OpenAlex
- https://openalex.org/W39858224
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:81007043
Keywords
Immune system, Medicine, Immunology, Autoimmune disease, Autoimmunity
References
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- Lipopolysaccharide induces sickness behaviour in rats by a vagal mediated mechanism.
- Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis.
- Influence of adrenaline on the dissemination of antibody-producing cells from the spleen.
- Innervation of the synovial membrane of the cats joint capsule
- Adrenergic receptors on monocytes modulate complement component synthesis.
- Elevated sympathetic nervous system activity in patients with recently diagnosed rheumatoid arthritis with active disease.
- Lymphocyte migratory pathways in adjuvant disease. I. Distribution of 51Cr-labeled thoracic duct lymph-borne.
- LYMPHATIC DRAINAGE AND ADJUVANT-INDUCED ARTHRITIS IN RATS.
- Lymphocyte blast transformation. I. Demonstration of adrenergic receptors in human peripheral lymphocytes.
- Modulatory effects of glucocorticoids and catecholamines on human interleukin-12 and interleukin-10 production: clinical implications.
- Potentiation of lymphokine-activated killer cell differentiation and lymphocyte proliferation by stimulation of protein kinase C or inhibition of adenylate cyclase.
- Regulation of tumor necrosis factor production by adrenaline and beta-adrenergic agonists.
- Quantitative analysis of cytokine gene expression in rheumatoid arthritis.
- Receptor-Mediated Interaction Between the Sympathetic Nervous System and Immune System in Inflammation
- The heterogeneity of mononuclear phagocytes in lymphoid organs: distinct macrophage subpopulations in rat recognized by monoclonal antibodies ED1, ED2 and ED3.
- The paraventricular nucleus of the hypothalamus and the functional neuroanatomy of visceromotor responses to stress.
- Catecholamines modulate human NK cell circulation and function via spleen-independent beta 2-adrenergic mechanisms.
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