ncRNA-mediated SOX4 overexpression correlates with unfavorable outcomes and immune infiltration in hepatocellular carcinoma
Explore this paper's citation graph
Summary
It is indicated that ncRNA-mediated upregulation of SOX4 correlated with the immune infiltration level and poor prognosis in HCC, which provides new directions for the development of novel immunotherapeutic targets for HCC.
- Type
- article
- Published
- 2024-08-14
- Cited by
- 3
- References
- 43
- OpenAlex
- https://openalex.org/W39143462
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:271871442
Keywords
Geography
References
- Treatment strategy for hepatocellular carcinoma in China: radiofrequency ablation versus liver resection.
- Tumor‐infiltrating lymphocytes for the treatment of metastatic cancer
- The unexpected role of lymphotoxin β receptor signaling in carcinogenesis: from lymphoid tissue formation to liver and prostate cancer development
- The role of TNF and Fas dependent signaling in animal models of inflammatory liver injury and liver cancer.
- Growth inhibition of hepatocellular carcinoma tumor endothelial cells by miR-204-3p and underlying mechanism.
- T cell exhaustion
- IL-6 pathway in the liver: From physiopathology to therapy.
- Immunology in the liver — from homeostasis to disease
- miR‐204‐5p targeting SIRT1 regulates hepatocellular carcinoma progression
- Predictive biomarkers for checkpoint inhibitor-based immunotherapy
- Regulatory T cells in cancer immunotherapy
- Promising significance of the association of miR-204-5p expression with clinicopathological features of hepatocellular carcinoma
- UALCAN: A Portal for Facilitating Tumor Subgroup Gene Expression and Survival Analyses
- TIMER: A web server for comprehensive analysis of tumor-infiltrating immune cells
- Immune Responses in the Liver.
- Immune checkpoint therapy in liver cancer
- Precision immunoprofiling by image analysis and artificial intelligence
- Determining consistent prognostic biomarkers of overall survival and vascular invasion in hepatocellular carcinoma
- CD8 T Cell Exhaustion During Chronic Viral Infection and Cancer.
- MiR-204 inhibits hepatocellular cancer drug resistance and metastasis through targeting NUAK1.
Cited by
Related papers
No related papers recorded.