Essential role of NLRP3 and AIM2 inflammasomes in IL-1beta production induced by the extracellular pathogen, enterohemorrhagic Escherichia coli
Explore this paper's citation graph
- Type
- article
- Published
- 2011-04-01
- Cited by
- 0
- References
- 0
- OpenAlex
- https://openalex.org/W33215702
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:81881714
Keywords
Escherichia coli, Microbiology, Pathogen, AIM2, Extracellular
References
Cited by
No citing papers recorded for this paper.
Related papers
- Enteropathogenic Escherichia coli Uses NleA to Inhibit NLRP3 Inflammasome Activation
- Modulation of the Inflammasome Signaling Pathway by Enteropathogenic and Enterohemorrhagic Escherichia coli
- Diverse mechanisms for inflammasome sensing of cytosolic bacteria and bacterial virulence.
- Effector triggered manipulation of host immune response elicited by different pathotypes of Escherichia coli
- Type VI secretion system contributes to Enterohemorrhagic Escherichia coli virulence by secreting catalase against host reactive oxygen species (ROS)
- Mechanosensing regulates virulence in Escherichia coli O157:H7
- Global Regulator of Virulence A (GrvA) Coordinates Expression of Discrete Pathogenic Mechanisms in Enterohemorrhagic Escherichia coli through Interactions with GadW-GadE
- Regulation of hemolysin in uropathogenic Escherichia coli fine-tunes killing of human macrophages