Human subiculo-fornico-mamillary system in Alzheimer’s disease: Tau seeding by the pillar of the fornix
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Summary
It is shown that non-fibrillary, hyperphosphorylated Tau is transported by the axons of the PoF from the subiculum to the mamillary body and has a seeding activity.
- Type
- article
- Published
- 2019-12-10
- Cited by
- 23
- References
- 70
- OpenAlex
- https://openalex.org/W31822997
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:208957664
Keywords
Unconscious mind, Psychology, Psychoanalysis
References
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Cited by
- Pyk2 Overexpression in Postsynaptic Neurons Blocks Aβ1-42-induced Synaptotoxicity in a Microfluidic Co-Culture Model
- Neurofibrillary changes undergoing morphological and biochemical changes – How does tau with the profile shift of from four repeat to three repeat spread in Alzheimer brain?
- Pyk2 overexpression in postsynaptic neurons blocks amyloid β1–42-induced synaptotoxicity in microfluidic co-cultures
- Seeding-Competent Tau in Gray Matter Versus White Matter of Alzheimer’s Disease Brain
- Clinical and neuropathological diversity of tauopathy in MAPT duplication carriers
- Ad memoriam : Charles Duyckaerts
- Deep brain stimulation of fornix in Alzheimer's disease: From basic research to clinical practice
- Early and selective subcortical Tau pathology within the human Papez circuit
- Deep brain stimulation for Alzheimer’s disease – current status and next steps
- Altered fornix integrity is associated with sleep apnea‐related hypoxemia in mild cognitive impairment
- The influence of APOEε4 on the pTau interactome in sporadic Alzheimer’s disease
- Amyloid-β peptide signature associated with cerebral amyloid angiopathy in familial Alzheimer’s disease with APPdup and Down syndrome
- Two distinct cell types of the medial mammillary body forming segregated subcircuits
- A dorsal subiculum-medial mammillary body pathway for spatial memory
- Impairments of spatial memory retrieval via medial mammillary body dysfunction in Alzheimer’s disease model
- The Determinant of Tau Spreading in Alzheimer’s Disease: Dependent on Senile Plaque, Neural Circuits, or Spatial Proximity?
- Proteomic remodelling of the neurofibrillary tangle from “PART” to advanced Alzheimer’s disease
- Professor Charles Duyckaerts (1951–2022)
- Hypothalamic microstructure and function are associated with mild cognitive impairment in aging.
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