Pyroptosis by Caspase11/4-Gasdermin-D Pathway in Alcoholic Hepatitis
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Summary
These results implicate pyroptosis induced by the CASP11/4‐GSDMD pathway in the pathogenesis of AH.
- Type
- article
- Published
- 2018-02-27
- Cited by
- 203
- References
- 45
- Access
- Open access
- OpenAlex
- https://openalex.org/W29108122
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:4962574
Keywords
MOX fuel, Burnup, Plutonium, Nuclear engineering, Radiochemistry
References
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- Caspase-11 cleaves gasdermin D for non-canonical inflammasome signalling
- Neutralization of IL-18 Reduces Neutrophil Tissue Accumulation and Protects Mice Against Lethal Escherichia coli and Salmonella typhimurium Endotoxemia1
- IL-1 receptor antagonist ameliorates inflammasome-dependent alcoholic steatohepatitis in mice.
- Supplementation of Saturated Long-chain Fatty Acids Maintains Intestinal Eubiosis and Reduces Ethanol-induced Liver Injury in Mice
- Interleukin-18 Facilitates the Early Antimicrobial Host Response to Escherichia coli Peritonitis
- Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury
- Th17 cells and their associated cytokines in liver diseases
- Pyroptotic cell death defends against intracellular pathogens
- Mouse intragastric infusion (iG) model
- Early liver transplantation for severe alcoholic hepatitis.
- Carbon monoxide from heme catabolism protects against hepatobiliary dysfunction in endotoxin-treated rat liver.
- Mouse model of chronic and binge ethanol feeding (the NIAAA model)
- Liver progenitor cell markers correlate with liver damage and predict short‐term mortality in patients with alcoholic hepatitis
- Enteric Dysbiosis Associated with a Mouse Model of Alcoholic Liver Disease
- Expression and alternative processing of IL‐18 inhuman neutrophils
- Betaine decreases hyperhomocysteinemia, endoplasmic reticulum stress, and liver injury in alcohol-fed mice.
Cited by
- Cell death in drug-induced liver injury.
- Pyroptosis, a Novel Player for Alcoholic Hepatitis?
- Pyroptosis: An inflammatory link between NAFLD and NASH with potential therapeutic implications
- Complement Factor D protects mice from ethanol-induced inflammation and liver injury.
- Hepatocytes and neutrophils cooperatively suppress bacterial infection by differentially regulating lipocalin-2 and NETs
- Impaired TFEB-mediated Lysosome Biogenesis and Autophagy Promote Chronic Ethanol-induced Liver Injury and Steatosis in Mice
- Current Understanding of Alcoholic Liver Disease: A prevalent "hidden" disease of increasing clinical importance
- A Unifying Hypothesis Linking Hepatic Adaptations for Ethanol Metabolism to the Proinflammatory and Profibrotic Events of Alcoholic Liver Disease
- Summary of the 2018 Alcohol and Immunology Research Interest Group (AIRIG) meeting
- Inflammasome: A Double-Edged Sword in Liver Diseases
- CX3CR1 differentiates F4/80low monocytes into pro-inflammatory F4/80high macrophages in the liver
- The Possibility and Molecular Mechanisms of Cell Pyroptosis After Cerebral Ischemia
- Exogenous nanoparticles and endogenous crystalline molecules as danger signals for the NLRP3 inflammasomes
- RIP Kinases in Liver Cell Death, Inflammation and Cancer.
- Glycyrrhizin attenuates hepatic ischemia‐reperfusion injury by suppressing HMGB1‐dependent GSDMD‐mediated kupffer cells pyroptosis
- Acute-on-chronic liver failure in patients with alcohol-related liver disease.
- Inflammatory Pathways in Alcoholic Steatohepatitis
- The non-transcriptional activity of IRF3 modulates hepatic immune cell populations in acute on chronic ethanol administration in mice
- The relevance of pyroptosis in the pathogenesis of liver diseases
- Cell Death and Liver Disease
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