Current hypotheses on the mechanisms of alcoholism
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Summary
Overall chronic ethanol-induced neuroimmune gene induction is proposed to alter limbic and frontal neuronal networks contributing to the development and persistence of alcoholism.
- Type
- article
- Published
- 2014-01-01
- Cited by
- 56
- References
- 229
- Access
- Open access
- OpenAlex
- https://openalex.org/W25307591
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:4853038
Keywords
Geography
References
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Cited by
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- IL-1 receptor signaling in the basolateral amygdala modulates binge-like ethanol consumption in male C57BL/6J mice
- Mechanisms of neuroimmune gene induction in alcoholism
- Alcohol consumption induces global gene expression changes in VTA dopaminergic neurons
- The potential role of amygdaloid microRNA-494 in alcohol-induced anxiolysis
- Adolescent Development of Cortical and White Matter Structure in the NCANDA Sample: Role of Sex, Ethnicity, Puberty, and Alcohol Drinking.
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- Chronic intermittent ethanol exposure leads to alterations in brain-derived neurotrophic factor within the frontal cortex and impaired behavioral flexibility in both adolescent and adult rats
- Nalmefene Prevents Alcohol‐Induced Neuroinflammation and Alcohol Drinking Preference in Adolescent Female Mice: Role of TLR4
- Persistent Decreases in Adult Subventricular and Hippocampal Neurogenesis Following Adolescent Intermittent Ethanol Exposure
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