Are postnatal hemangioblasts generated by dedifferentiation from committed hematopoietic stem cells?
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Summary
It is suggested that postnatal hemangioblasts are generated by dedifferentiation of committed hematopoietic stem cells and that gene transcription exists in reversible "fluctuating continua" during parental cell cycles.
- Type
- article
- Published
- 2007-05-01
- Cited by
- 19
- References
- 247
- OpenAlex
- https://openalex.org/W17577919
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:39574375
Keywords
Computer science
References
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- The presence of JAK2V617F mutation in the liver endothelial cells of patients with Budd-Chiari syndrome.
- Reversible and Noisy Progression towards a Commitment Point Enables Adaptable and Reliable Cellular Decision-Making
- An adult uterine hemangioblast: evidence for extramedullary self-renewal and clonal bilineage potential.
- Spleen endothelial cells from patients with myelofibrosis harbor the JAK2V617F mutation.
- Primitive and definitive erythropoiesis in the yolk sac: a bird's eye view.
- The Generation of Programmable Cells of Monocytic Origin Involves Partial Repression of Monocyte/Macrophage Markers and Reactivation of Pluripotency Genes
- Circulating angiogenic monocyte progenitor cells are reduced in JAK2V617F high allele burden myeloproliferative disorders.
- The role of bone-marrow-derived cells in tumor growth, metastasis initiation and progression.
- Final checkup of neoplastic DNA replication: evidence for failure in decision-making at the mitotic cell cycle checkpoint G(1)/S.
- Expression of Epidermal Growth Factor Receptor by an Experimental Murine Tumor of Acute Myeloid Leukemia Origin
- Definitive erythropoiesis in chicken yolk sac
- Mutational Heterogeneity in Human Cancers: Origin and Consequences
- Non-hematopoietic Stem and Progenitor Cells Derived From Human Umbilical Cord Blood
- 肿瘤DNA复制的最终检查:在有丝分裂细胞周期检查点G1 / S上决策失败的证据
- The presence of JAK 2 V 617 F mutation in the liver endothelial cells of patients with Budd-Chiari syndrome
- MYELOID NEOPLASIA Spleen endothelial cells from patients with myelofibrosis harbor the JAK2 V617F mutation
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